Aperson'strait,thinking or behaviour is classified as abnormal if it is rare or statisticallyunusual. Any unusual behaviour will be more than two standard deviations from the mean, that is, it will be found in less than 5% of the population
When a person can nolongercope with the ordinarydemandsofeverydaylife. This might be a lack of hygiene, poor nutrition, unable to work or can not maintain a relationship
Behavioural: Panic-crying, screaming, running away or freezing (fainting), flight/fight, Avoidance-effort to keep away from the phobic stimulus. Making it hard to go about daily life. E.g. public toilet fear (can't go out), Endurance-in unavoidable situations (i.e. flying) continuous and extreme anxiety
Emotional: Excessive fear and an unpleasant state of high arousal. Prevents sufferer relaxing and cannot experience positive emotion. Fear is immediate and extremely unpleasant response we experience when we encounter or think about the phobic stimulus
Cognitive: Irrational thoughts. Person knows that their fear is excessive. Thinking resists rational arguments about the phobia and they find it difficult to focus their attention elsewhere, known as selective attention
Behavioural: Low levels of energy, lethargic (withdrawn from work, social life) in extreme cases they can't get out of bed, Psychomotor agitation-struggle to relax (pace up and down), Disruption to sleep (insomnia or hypersomnia), Appetite increase or decrease-affecting weight, Verbal aggression (ending a relationship or Job) or physical aggression (self-harm, suicide)
Emotional: Lowered mood (worthless, empty), Anger (at self or others) may lead to self-harming, Self-esteem is low (self-loathing)
Cognitive: Poor levels of concentration (unable to stick with a task), Pay more attention to negative aspects of a situation and ignore positives, Tend to recall unhappy events rather than happy ones
Behavioural: Compulsions: compelled to repeat a behaviour and reduce anxiety, Coping strategies: praying - but can be distracting
Emotional: Characterised by anxiety-irrational fear (compulsions bring temporary relief), Can also experience depression
Cognitive: Obsessions: Intrusive thoughts-RECURRING over again, (i.e.. Being contaminated by dirt, door unlocked get hurt), Selective attention towards their anxiety
Classical Conditioning - Little Albert, Operant Conditioning - The behaviour is strengthened when an unpleasant consequence is removed. The sufferer avoids the anxiety by avoiding the situation which negatively reinforces their phobia
The behaviour is strengthened when an unpleasant consequence is removed. The sufferer avoids the anxiety by avoiding the situation which negatively reinforces their phobia.
Seligman argues that phobias can develop without a traumatic event. He argues that animals including humans are genetically programmed to learn an association between dangerous stimuli & fear. Things that would be dangerous in the evolutionary past are more common than modern fears.
1. Stage One: The client is taught relaxation techniques.
2. Stage Two: Construct a hierarchy of fear.
3. Stage Three: The client works through each stage, using the relaxation techniques at every step. Only when the client can achieve the step and not feel anxious do they move up to the next one.
The client is firstly taught relaxation techniques but then they are exposed to their fear for a long period time and in it's most fearful form. As adrenaline naturally decreases, a new stimulus-response link can be learned between the stimuli and relaxation.
Systematic desensitisation is 75% effective when treating phobias (McGarth, 1990). Choy et al (2007) reported that flooding and SD were effective but that flooding was the more effective of the two in treating phobias.
Flooding is an unethical treatment, as it can be highly distressing for the individual and one could argue that their right to withdraw is denied to them during the exposure.
B-Belief. For depression, these beliefs are irrational.
C-Consequences. Rational belief's lead to healthy emotions and the ability to have a 'normal' life. Irrationalbeliefs lead to unhealthy emotions and could develop into depression.
Beck's Depression Inventory (BDI) allows us to assess depressive symptoms in patients and develop cognitive therapies. Ellis developed REBT which can challenge irrational beliefs and relieve depressive symptoms.
Boury et al (2001) monitored students negative thoughts with Becks BDI and found that depressives misinterpret facts and experiences in a negative fashion and feel helpless about the future.
Some say that the cognitive approach blamesthepatient for their depression, as it fails to acknowledge otherinfluences other than theirownirrationalthoughts.
It aims to challengeirrationalanddysfunctionalthoughtprocesses by identifying and testing the negative beliefs. They may have homework to complete in which they recordpositiveevents.
Effectivedisputing can change the way we think about things and allow for more rational thought processes. It adds DEF steps to the ABC model: D = Dispute & Effect and F Feelings. Different types of disputing use logic, empirical evidence and pragmatic disputing (is this thought useful to me?)
March et al. (2007) compared CBT with drugs and a combination of the two in 327 adolescents. 81% of CBT improved, 81% of drug group improved and 86% of combination improved. CBT is just as effective as antidepressants and works well alongside drugs.
Cognitive therapy is time consuming and expensive and there can be waiting lists. Relapse rates can also be high in the long term. 53% within a year relapse (Shehzad Ali et al., 2017)
It can't be used for severe cases and so the patient must wait until drugs kick in and which make them more alert and motivated to take part.
Lewis (1936) found that 37% of his OCD patients had parents with OCD and 21% had siblings with OCD. Suggesting OCD runs in families. The COMT gene regulates the production of dopamine and high levels of dopamine might be the cause of OCD. Mutations of the SERT gene affects the transport of serotonin, creating lower levels of the neurotransmitter. These lower levels are also implicated in OCD.
It is thought that dopamine levels are abnormally high and/or serotonin levels are low in people with OCD. When the caudate nucleus is damaged it fails to suppress the 'worry' signals and the thalamus is alerted constantly.
Twin Studies: Nestadt (2000) reviewed twin studies and found 68% of monozygotic twins shared OCD as opposed to 31% of dizygotic, demonstrating a genetic cause.
Concordance Rates: Concordance rates are never 100%. The diathesis-stress model may be a better explanation, where we have a predisposition to OCD but environmental factors determine if we get it.
Animal Studies: Szetchman et al (1998) found if he increased rats' dopamine levels with drugs, they would display stereotypical behaviours resembling the compulsive behaviours found in OCD sufferers.
SSRI's (selective serotonin reuptake Inhibitors) are most commonly used for OCD. Drugs such Zoloft, Praxil and Prozac increase levels of serotonin in the synapse which reduces symptoms. If SSRIs are not effective after 3-4 months other drugs may be tried such as Tricyclics, which have the same effect on serotonin as SSRIs but have more severe side effects or SNRIS which increase serotonin and noradrenaline.
Effectiveness:Soomro (2008) reviewed 17 studies where SSRI's had been used to treat OCD and found in some cases the SSRIwasmoreeffectivethanaplacebo in reducing the symptoms of OCD.
SideEffects: SSRI's can cause headaches,nausea and insomnia which can mean that people stoptakingthemedication. The side effects of Tricyclics are more severe, including hallucinations and irregular heart beat and weight gain.
CostEffectiveness: Drug therapies are relativelycheap when compared to psychological therapies. They also do not require much motivation from the patient to take them unlike cognitive therapies. However, they do only treat the symptoms of OCD not the root cause. If the patient stops taking them then the symptoms usually return.