Born before completion of 37 weeks of gestation regardless of birth weight
Gestational age categories
Late preterm (34 0/7 through 36 6/7)
Early term (37 0/7 through 38 6/7)
Full term (39 0/7 through 40 6/7)
Late term (41 0/7 through 41 6/7)
Postterm (42 0/7 and beyond)
Postmature
Born after completion of 42 weeks and showing the effects of progressive placental insufficiency
Fetal growth categories
AGA (appropriate for gestational age)
LGA (large for gestational age)
SGA (small for gestational age)
Immediate interventions
1. Airway maintenance
2. Maintaining adequateoxygen supply
3. Maintaining bodytemperature
4. Initiating nutrition
5. Promoting parent infant interaction
Newborn medications
Eye prophylaxis to prevent opthalmia neonatroum or neonatal conjunctivitis
Vitamin K prophylaxis
Hep B vaccine
Birth injuries
Retinal and subconjunctival hemorrhage
Soft tissue injuries like erythema ecchymosis petechiae
Trauma secondary to dystocia
Accidental lacerations
Hypoglycemia
Usually defined as blood glucose less than45, infants at risk are preterm or late preterm SGA or LGA low birth weight infants of mothers with diabetes infants who experience perinatal stress such as asphyxia cold stress or respiratory distress
Pathologic jaundice
Disorders exacerbate physiologic processes that lead to hyperbilirubinemia, onset < 24 hrs of age, levels elevate faster and jaundice last longer, more dangerous
Physiologic jaundice
Increased bilirubin reabsorption due to delayed bilirubin excretion because of delayed metabolism, frequent and long feedings early are primary prevention
Breastfeeding-associated jaundice
Breastfeeding doesn't cause jaundice rather it is the lack of effectivebreastfeeding that contributes to the hyperbilirubinemia, breastfeeding associated jaundice is early onset and it isn't the breastfeeding but rather the poor breastfeeding that is the problem, breast milk jaundice late onset jaundice and something is wrong with the milk and recommended to stop breastfeeding
Causes of unconjugated hyperbilirubinemia
Rh/ABO incompatibilities
Infections
Inherited RBC disorders
Bruising or internal bleeding
Hemangiomas
Hypoxia or asphyxia insults
Polycythemia
Swallowed blood with delivery
Delayed feeds and passage of meconium
Causes of conjugated hyperbilirubinemia
Neonatal hepatitis
Intestinal obstruction
Ischemic necrosis of liver
Parenteral alimentation (tube feedings)
Metabolic disorders
Hematologic disorders
Ductal disorders
Biliary atresia
Tumors of liver and biliary tract
Hyperbilirubinemia
When bilirubin exceeds the levels that can be bound and metabolized and it starts to build up in the blood, bilirubin freely crosses the blood brain barrier and at high levels can cause toxicity, acute bilirubin encephalopathy is acute manifestations of bilirubin toxicity, kernicterus is irreversible long term consequences yellow brain from not treated promptly
Assessing hyperbilirubinemia
Blanch the skin in natural daylight or observe the sclera, it progresses head to toe, blood sampling with lab draw at 24 hours of age and repeated as indicated by values
Treating hyperbilirubinemia
Feedings early and often 8-12hrs/24hrs, phototherapy, blue lights because phototherapy converts bilirubin to photobilirubin which can be excreted in urine bypassing need for liver conjugation, exchange transfusions 85% of neonatal RBCs replaced by donor cells but only in NICU
Universal newborn screening
Mandated by US law usually at 24 hours, early detection of genetic diseases that result in severe health problems if not treated early, newborn hearing screening, screening for critical congenital heart disease (O2 sat on right arm and right leg, results must be greater than 95% and within 3% of each other to pass)
Intramuscular injections
Always in the leg for immunizations and medications
Circumcision
Policies and recommendations, parental decision, procedure either yellen (Gomco) or Mogen clamp or plastibell device, pain management with sucrose, care of the newly circumcised infant, infection and bleeding main complications