Involved in personality expression, decision making (executive functioning), moderating social behaviour (inhibiting and allowing behaviour), choosing between "right" and "wrong" (conscience), long-term planning
Brain regions we are building
Hippocampus
Amygdala
Prefrontal Cortex
Broca's Area (language production)
Wernicke's Area (language comprehension)
Arcuate Fasciculus (large bundle of nerves)
Arcuate Fasciculus
Large bundle of nerves (Latin for Curved Bundle)
Akinetopsia
Motion blindness, seeing the world in "still shots" (stroboscopic)
V5/MT
Visual area responsible for the perception of motion, damaged in Ms. Leibold causing Akinetopsia
The Visual System
Parallel Processing - a series of neurons communicate information from the retina to the cortex
Visual Processing
1. Photoreceptors (rods and cones) on the retina
2. Bipolar cells transmit signals
3. Ganglion cells (optic nerve)
Fovea
Concentration of cones
Visual Processing
Bipolar cells transmit signals to Ganglion Cells (Optic Nerve)
Contralateral & Ipsilateral Paths
Paths of visual information from the eyes to the brain
Lateral Geniculate Nucleus
Inside the Thalamus, contains Parvocellular Cells (size, color, shape/form analysis, critical for faces) and Magnocellular Cells (motion and depth, guides directed movement)
Parvocellular Cells
Begin processing "what"
Magnocellular Cells
Begin processing "where"
Primary Visual Cortex (V1)
Also called the Striate Cortex, at the very back of the occipital lobe, highly specialized for processing information about static and moving objects, excellent at pattern recognition/differentiation
Dorsal (Where) Stream
Location of objects and guiding of our responses/movements (e.g. reaching, picking up a glass, catching a ball), Occipital-parietal pathway (up to the Sensory Cortex & Motor Cortex)
Ventral (What) Stream
Identification of objects and faces, Occipital-temporal pathway (connects to Limbic System – contains the Hippocampus and the Amygdala)
Visual Agnosias
Caused by disruptions to the What pathway, including Prosopagnosia (inability to recognize faces) and Visual Agnosia (inability to recognize objects)
Capgras Delusion
No connection of face to emotion center, visual recognition without emotional recognition
Associative agnosia
Can see the object but cannot describe it from sight, nor name it, do not know what it is. Cannot connect to previous knowledge.
Lateral Inhibition
In the ganglion cells (Optic Nerve), some excited cells can inhibit the excitement (firing rate) of their neighbors on one or both sides, decreasing their neighbors firing rates
Apperceptive agnosia
Can only see an objects shapes, colors and position (features) but cannot see them together. i.e. Cannot see intact objects. They cannot look at an object and draw it. They can draw well from memory
Less intense stimuli (darker areas)
Result in less lateral inhibition, edge looking darker against a lighter edge
Bottom-up (or data-driven) processing
Stimulus-driven effects. Features of objects - lines, curves etc. From parts to the whole.
Lateral Inhibition
Helps define an object's shape through edge enhancement - the lighter edge appears brighter and the darker edge beside it appears even darker due to contrast
Top-down (or concept-driven) processing
Knowledge- or expectation-driven effects. Knowing what something "should" be - based on previous knowledge and inference. From what the expected whole is down to its individual parts (features).
Center-surround Cells
Aka Dot Detectors
Features
Building blocks. Things various objects have in common. Play a role in visual search
Edge Detectors
Cells that show preferential firing for certain edge orientations (horizontal to vertical)
Integrative agnosia
Symptoms of both apperceptive and associative agnosia. Impaired in seeing objects as wholes - cannot judge how the features are bound together. This suggests that we perceive things in parts - features that we then bind together somehow.
Visual Cells
Some cells fire maximally at certain sized angles
Some fire maximally in response to corners or notches
Some cells fire in response to specific types of movement (e.g. left to right, vertical from up to down)
Parallel processing splits up visual processing, but we do not see the world as disjointed - this is the binding problem
Tachistoscope
A word or a list of words is presented to the participant for a controlled brief amount of time (20 - 40 milliseconds) each. Between each presentation of a word, a mask is presented. The mask, a collection of letters, is intended to disrupt rehearsing the word - prevents any continued processing. Then they are asked if they saw certain words. Recognition
Elements that help solve the binding problem
Spatial position - each processing location has a map based on where everything is in the visual field
Neural synchrony - the brain keeps track of when the various neurons fire in the different brain areas to establish a pattern of what features go with what
Repetition priming
Words that have appeared recently to the participant are more easily recognized.
Attention is critical for binding visual features together, when attention is overloaded people will make conjunction errors - errors in binding
Conjunction Error
An error in binding visual features together
Word-superiority effect
People's response when asked whether "DARK" had an "E" or a "K" is fastest. Also faster than when a letter is presented within a nonsense letter string such as "JPHKW."
3 systems that govern attention
Orienting System
Alerting System
Executive System
Degree of Well-Formedness
How statistically likely it is for a specific letter combination to occur. E.g - THE is extremely likely to occur & always primed.
Orienting System
Responsible for disengaging attention, shifting it, and reengaging on a new target