sudden onset chest pain is caused by PE and heart attact
some sign of cardiac failure include: orthopnea, paroxysmal nocturnal dyspnea and no chest pain
low bp and congestive cardiac failure are cardiogenic shock
if the pulse is regular,then there is no arrhythmia
if patient is in shock, they are at high risk of cardiac failure
holosystolic murmur/pansystolic is a systolic murmur caused by mitral regurgitation, tricuspid regurgitation, aortic stenosis and pulmonary stenosis
lead ii, iii, avf are inferior part of the heart supplied by branch of RCA-posterior descending artery
prednisone is a corticosteroids.
hydroxycloroquine is an antimalarials
patient experiencing chest pain during exersion and the pain goes away, think about angina
no crackles meaning no congestive heart failure, no rash-exclude rheumatic heart disease
remember HTN patients needs to be on life style modification first before we can increase medication dose
always give diuretic for congestive heart failure,
severe edema in heart failure, give spironolactone
severe hypertension is associated with >180/110 but asymptomatic
hypertension urgency is associated with: >180/110 plus headache and epistaxis(nose bleed) OR retinopathy grade1-artery thickening, grade 2-vein constriction where they meet arteries
hypertension emergency/hypertension crisis/malignant hypertension is associated with >180/110 plus signs of end-organ damage e.g Brain damage- seizures,confusion,CVA-there is subarachnoid hemorrhage. management is to slowly decrease bp over 48hrs. give hydralazine and labetolol
If patient has MI, but there is ST depression on ecg, this is NSTEMI.
patients with acute MI can develop mitral regurgitation 2-7 days after having infarction.And mitral regurgitation lead to diastolic overload of ventricles meaning there is an in increase in left ventricular filling pressure.
positive stress test is associated with st depression and t-wave inversion, next test will be echocardiogram or coronary angiogram
for hypertension patients we check compliance, their lifestyle, then we can maximize dose then they can comeback after a week
in wet-cardiac failure(congestive heart failure) do not give beta blocker
retinopathy grade3- retinal ischaemia and grade4- papilloedema
ACE-inhibitor side effect is angioedema, which is swelling of the face, lips, tongue, and throat
respiratory distress signs include: wheezes, grunting(niosy breathing sound),retractions(area between ribs and neck deepens/sink in when they inhale),nasal flaring(nostrils widen while breathing),cyanosis, tachypnea, sweating
pleural disease is associated with pleural chest pain that gets worse on inhalation and become better when patient leans forward.
pulmonary hypertension signs include: palpable and loud second heart sound(P2),parasternal heave (heart pulse palpable) plus cardiac failure
differential diagnosis of chest pain include: coronary artery disease, pulmonary or pleuritic(pleurizy,pneumonia,pericarditis,PE), aortic(dissection,intramural hematoma), gastrointestinal/esophageal, chest wall/musculoskeletal.
chest pain coronary artery disease is: 1)substernal pain, 2)pain radiate to arm-shoulder or jaw,3)pain gets worse with exertion and relieved by rest or nitroglycerine.
pulmonary/pleuritic chest pain: 1) sharp/stabbing pain, 2)pain gets worse with inhalation,3)pericarditis chest pain gets worse when lying flat,4)PE and pneumothorax they have hypoxia and respiratory distress
aortic dissection or intramural hematoma chest pain is: 1)sudden severe(tearing)pain,2)radiates to the back,3)common in elderly men,4)patient have hypertension and risk factors for atherosclerosis
gastrointestinal/esophageal chest pain is: 1)nonexertional and pain is relieved by antacids, 2)pain is substernal or upper abdominal, 3)pain is associated with regurgitation,nausea,dysphagia(difficulty swallowing), 4)nocturnal pain
chest wall/musculoskeletal chest pain is: 1)persistent prolong pain, 2)pain gets worse with movement
antiplatelets include: prasugrel and clopidogrel
patients with ventricular tachycardia but they are unstable, we take them straight to sychronized cardioversion(electrical cardioversion).
if the patient with ventricular tachycardia is stable,we need to do conservative management; 1) carotid massage-if is not effective, 2) do valsalva maneuver-if not effective,3) medication-give antiarrhythmic(Adenosine or Amiodarone)-if not effective,4) synchronized cardioversion
panic attack= tachycardia
if patient is unstable and there is no pulse, we take the patient to unsynchronized cardioversion(defibrillation)
COPD plus cardiac failure cause core pulmonale which is pulmonary hypertension