W: culture-bound: self-actualise, self—indulgent in cultures, high indepenfence good in germany, bad in italy
W: ‘ideal’ mental health impossible to maintain all the time HOWEVER practical value to someone wanting to improve mental health
abnormality - deviation from social norms
social norms: collective judgement as a society, specific to a culture - difficult to globalise
S: application: antisocial personality disorder defined by failure to conform to social norms + schizotyal perosnailty disorter uses ‘strange’ to characterise thinking
W: cultural + situational relativism: hearing voiced + being decietful normal in some cultures/ situation but not all
W: humanrightsabuse: nymphomania used to control women HOWEVER need deviation for specificdiagnosis
abnormality - statistical infrequency
how often you come across it (unusual = abnormal)
68% score 100 IQ (normal), 2% score below 70 - abnormal + diagnosis of intellectual disability disorder
S: link to phobias: DeJonah et al - 73% dental phobia experienced related trauma - control group only 21% HOWEVER not all phobias come from trauma + not all trauma leads to phobia
credible individual explanation HOWEVER general aspects better explained by evolutionary theory (fear of dark)
W: traumatic: Schumacher et al - flooding rated more stressful than SD - ethical issues + high attrition rates HOWEVER informed consent achieved before hand
W: doesn’t tackle cause: Pearson case study: woman with phobia of death cured then developed phobia of being criticised
S: March et al: 327 adolescents, after 36 weeks: 81% CBT, 81% drugs, 86% both improved - just as effective as others HOWEVER effectiveness: severe cases unmotivated (HOWEVER Lewis + Lewis as effective as drugs + behavioural therapies) + Sturmey - any form of psychotherapy not good for learning disabilities -thinking (HOWEVER Taylor et al: if used appropriately, effective)
W: high relapse: Shehzad Ali et al - 439 people, 12 months of CBT - 42% relapse in 6 months, 53% a year
W: Yrondi et al: CBT least preferred psychological therapies
explaining OCD - genetics
faulty biological process - geneticvulnerability passed on
Lewis: 37% parents with OCD, 21% siblings
diathesis-stress model: certain genes leave you vulnerable to disorders (environmental stress triggers it) - candidate genes for OCD
COMT: transmission of dopamine
SERT: transmission of serotonin
5HT1-Dbeta: transport serotonin accross synapses
Taylor et al: OCD polygenic (cobimnation of genes) - 230 different genes for OCD
aetiologicallyheterogeneous: origins vary for every person
types of OCD result of particular genetic variations
explaining OCD - genetic evaluations
S: support: Nestadt et al: 68% of identical twins share OCD, 31% non-identical + Marini+ Stebnicki: 4x more likely to have OCD if family member has it - sone genetic influence
W: environmental risk factor: environment triggers/ increases risk of OCD - Cromer et al: over 1/2 OCD people experienced trauma - OCD more severy with more trauma - genetic vulnerability only partial explanation
W: difficult to find candidate genes HOWEVER Ahmari: studied mice and found particular genes involved in repetitive behaviour HOWEVER generalisability to humans is poor
explaining OCD -neural
genes associated with OCD effect levels of neurotransmitters + structures of the brain
serotonin (helps regulate mood) levels low
transmission of mood relevant information doesn’t take place
low mood + other mental processes
abnormal functioning of frontal lobes=impaired decision making related to cases of hoarding
left parahippocampal gyrus: processing unpleasant information functions abnormally with OCD
explaining OCD - neural evaluations
S: support: reduced OCD symptoms + Nestadt et al: symptoms form parts of conditions biological in origin (Parkinsons) - assume it biological processes underline OCD - biological factors responsible for OCD
W: serotonin not unique to OCD, confounding variable of depression - serpentine irrelevant to OCD
W: correlation vs causality: correlation or neural abnormality + OCD not necessarily a relationship - OCD may cause abnormal brain function or a 3rd factor responsible
treating OCD - drug therapy
selective serotonin reuptake inhibitors: prevent reabsorption + breakdown of serotonin, continues to stimulate postsynaptic neuron, more likelt ST binds to receptor
dosage varies with type
3-4 months daily use for an impact - no response: dosage increased of paired with other drugs (tricyclics or SNRIs)
combining SSRIs with other treatments: CBT (engage more effectively) or other drugs
treating OCD - drug therapy evaluations 1
S: effectiveness: Soomro et al - 17 studies compare SSRIs + placebo, 70% symptoms improve (30% improved with drugs/therapies) - drugs appear helpful HOWEVER not the most effective: Skapinakis et al - CBT therapies better - drugs not optimum
cost effective + non-disruptive: good use of limited NHS funds - take drugs until symptoms decline vs time in therapy
treating OCD - drug therapy evaluations 2
W: side effects: tricyclic domipromine - 1/10 erectionproblems + weightgain 1/100aggressive + heart problems - low quality of life - attrition rates
W: biased evidence: Goldacre - researchers sponsored by drug therapies + lack of independent studies HOWEVER best available evidence supports effectiveness
depression characteristics
behavioural: activitylevels,disruption to sleep + eating, aggression + self-harm