often followed by repetitive compulsions, impulses, urges
what is the ocd cycle?
obsessions - thoughts
anxiety - emotional
compulsions - behaviour/actions
relief - removinganxiety
2 behavioural characteristics?
compulsions
avoidance
explain compulsions?
repeatedlycarry out a behaviour
e.g washing yourself3/4 times to reduce anxiety
explain avoidance?
avoiding situations which cause obsessesion/compulsions
e.g not going to public toilets
3 emotional characteristics?
anxiety & distress
accompanyingdepression
guilt & disgust
explain anxiety & distress?
feel anxious if compulsion is not carried out & worrying something bad will happen
explain accompanying depression?
those that suffer from ocd comorbidly suffer from depression as they have a low mood
explain guilt & disgust?
guilty for their feelings & disgust in themselves/towards something else
3 cognitive characteristics?
obsessiveintrusive thoughts
cognitivestrategies to deal with obsessions
selectiveattention
explain obsessive thoughts?
having repetitive thought that they need to carry out a behaviour to prevent something bad from happening
explain cognitive strategies?
coming up with a way to deal with obsession
e.g praying/ritualistic thinking
explain selective attention?
directed towards the anxiety generating stimuli
biological explanation for ocd?
ocd is caused by physical/bodily problem
name 2 sub explanations of ocd?
genetic
neural - neurotransmitters & brain structure
explain neurotransmitters?
produced from brain
chemicals that transmit electrical messages from one nerve cell to another
suggests that ocd caused by imbalance in nt within brain
what is serotonin?
mood regulator
explain brain structure?
ocd caused by problems with structures of brain
several areas of frontal lobes are thought to be abnormal in ocd patients
person with ocd has high levels of activity in orbital frontal cortex
what is orbital frontal cortex(ofc)?
linked to carrying out an activity due to an impulse
explain genetics?
ocd may be inherited
this vulnerability may be passed on across generations
explain ocd is polygenic?
taylor 2013 found that there are up to 230 different genes that may be involved in disorder
explain ocd is aetiologically heterogenous?
there are number of different combinations of genes that can lead to disorder & this combination will vary between people
carey & gottesman (1981)?
found that monozygotic twins have concordance rate of 87%
dizygotic twins - 47%
shows that genetics must play role in ocd as those who are more genetically similar are more likely to share same disorder than those less genetically similar
what are candidate genes?
specific genes which mean people are more likely to suffer from ocd
explain sert gene?
serotonin
if mutated creates lower levels of serotonin in a person
explain comt gene?
dopamine - linked to pleasure & satisfaction
if too much - brain becomes saturated with dopamine & less responsive to pleasure
if too little - brains does not receive rewards it should get for completing a task
leads to brain needing to complete tasks repeatedly in order to feel satisfied
what is drug therapy?
works by altering amount of neurotransmitters in brain
what is ssri?
selective serotonin reuptake inhibitor
type of drug
e.g prozac - brand
what does ssri do?
increase amount of serotonin by blocking reuptake pump in synapse
allows more serotonin to attach to receptors as its not being taken away by pump
increases level of serotonin in brain which alleviates symptoms
2 * of drug therapy?
quick & easy to use
evidence that it is effective from soomro
* quick & easy?
patients can take drugs easily by swallowing a pill which is not as strenuous as other treatments such as cbt which requires patients to changethoughts
appropriate
easily accessible to anyone due to not being cog/phy demanding & is lesstimeconsuming
will increase likelihood of people engaging with this therapy - results in highersuccessrates
* evidence that drug therapy is effective?
soomro (2009) reviewed studies who compared patients treated with ssri & a placebo for ocd
found that in allstudiesssri was moreeffective
effective
evidence to support its success in treating patients compared to other methods
suggests there must be biocomponents to disorder as placebo effect had limitedsuccessratescompared to ssri which increase nt
3 X of drug therapy?
possible sideeffects
does not treat cause
not longlastingtreatment
X side effects?
drugs have unpleasantside effects such as nausea & insomnia which will affect dailyroutines of patients
? appropriate
there may be othertreatments which treatocd without causing these sideeffects so are more likely to be used by patients
explain X does not treat cause?
drugs are used only to increase levels of serotonin
means without drugs cognitive symptoms such as obsessions would still be there which lead to anxiety & compulsions
? effective
doesnt treat ocd fully onlyminimisessymptoms
means when patient stops taking drugdisorder will return increasingrelapserates
suggesting othertreatments may be more appropriate
X not long lasting treatment?
therapy does not guarantee a patient wont relapse as it is easy for patients using drugtherapy to do so
patient can simply stoptakingdrug at any time meaning ocdresurfaces due to lowerlevels of serotonin
? effective & appropriate
cannot be used as a suitabletreatment for ocdovertime & an alternativecognitive treatment might be more suitable
* evidence to support genetic from billet 1998?
found that mz twins were more than twice as likely to develop ocd than dz twins
valid
supports idea that genetics play a role in ocd as those who are more genetically similar are more likely to share conditions than those less genetically similar
X not 100% concordance for genetic exp?
suggests that ocd cannot be completely caused by genetics because if it was there would be 100% concordance rates between mz twins who share 100% genetics
? valid
suggests that there be other factors such as environmentalfactors that cause ocd