Coined the term 'double bind' to explain the contradictory situations children may be placed in by their parents.
- where verbal message given but opposite behaviour is exhibited
- parent tells child to be 'more spontaneous' but if child is more spontaneous it becomes confused and uncertain as by doing it they are clearly not being spontaneous.
---> leads to a negative reaction of social withdrawal and flat effect in order to escape double bind situations.
-theory was initially popular but Bateson was later accused of selective bias in focusing only on aspects of interviews with schizophrenics that supported his claims
- this and recent evidence supporting a genetic link has lessened support
Where families who persistently exhibit criticism and hostility exert a negative response especially on recovering schizophrenics who when returning to their families react to expressed emotions by relapsing to an active phase of the disorder and experience severe positive symptoms of hallucinations and delusions of persecution.
Levels of schizophrenia in adopted individuals who were the biological children of schizophrenic mothers was 5/8% in those adopted by healthy families compared with 36.8% for children raised in dysfunctional families.
-supports the idea that family dysfunction can play a role in the development of schizophrenia and that individuals with high genetic vulnerability are more likely to be affected by an environmental stressor (diathesis stress model)
Established 7 problems which characterise family dysfunction
- poor adult relationships
- lack of warmth between adults
- visible disturbance of mother - child relationship (f-c and sibling-c)
- parental overprotection and child abuse
Migrants who experienced at least 3 had 4 times the normal level of vulnerability compared to the double level risk of migrants not experiencing family dysfunction
- family dysfunction increases likelihood of stressors triggering schizophrenia
Reviewed 26 studies of expressed emotion, finding that the mean relapse of schizophrenia patients who returned to high expressed emotion families was 48% compared to 21% living with low expressed emotion families
- supports the idea that high expressed emotion increases the likelihood of relapse
Theory supported by successful therapies focusing on reducing expressed emotions within families and achieving lower relapse rates than other therapies.
theory can be said to have predictive validity as the theory must have some accuracy for the therapy to work
Focus on maladaptive thought processes as central feature of schizophrenia
- Beck and Rector (2005) proposed a cognitive model that combines a complex interaction of neurobiological, environmental, behavioural and cognitive factors to explain schizophrenia.
Abnormalities within brain functioning are seen as increasing vulnerability to stressful life experiences, which in turn lead to dysfunctional beliefs and behaviours
Occur where sufferers experience problems with attention, communication and information overload.
- sufferers are also seen as unable to deal with inappropriate ideas, such as misperceiving voices in their heads as people actually trying to speak to them, rather than perceiving them more sensibily as 'inner speech', which most people perceive
Delusions are seen as occurring because of active cognitive biases (thinking in irrational ways) such as external attributions like individuals believing they are being persecuted
- hallucinations are understood in terms of biased information processing, while cognitive deficits experienced by schizophrenics are referred to as alien control symptoms, where sufferers believe their thoughts and behaviours being influenced by external people and forces
Seen as occurring due to the use of cognitive strategies to control high levels of mental stimulation
- schizophrenics may actually experience a greater level of emotion than they physically display, as not displaying emotion is one strategy to control levels of emotion being experienced internally.
Reviewed available evidence to report that cognitive impairment is found in 75% of schizophrenics, particularly in memory, attention, motor skills, executive function and intelligence, supporting Beck and Rector's cognitive model. Cognitive impairments often pre-dated illness onset, did not occur as a result of substance abuse and were related to social and functional impairments.
Reported that schizophrenia is better characterised by cognitive deficits than symptoms and that these cognitive deficits are an enduring feature of schizophrenia which are not specific to subtypes of the disorder, with memory and attention being the main cognitive deficits forming the core dysfunction of the disorder
- supports the cognitive explanation of schizophrenia
- if true then it should be possible to construct a specific cognitive deficit profile from which to diagnose the disorder, this is yet to happen
Supported the work of Elvevag and Goldberg, finding that cognitive impairments are the core feature of schizophrenia
- they found that these impairments pre-date the onset of the disorder and are found throughout the course of it
- further support for beck and rector'scognitive model
- with additional support coming from the fact that they found that effective therapies seem to reduce cognitive deficits and thus improve functioning in schizophrenic patients
However, cognitivetheories in themselves do not explain what led to the cognitive dysfunctions seen in schizophrenics and therefore can't be seen as explaining the causes of schizophrenia .