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MSK 2
Midterm Exam
Craniovertebral Joint
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McKenzie Simpson
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Cards (36)
CV instability can result from:
trauma
RA
AS
corticosteroid
use
recurrent
upper respiratory tract infections
Grisel's
syndrome
spontaneous
AA dislocation
down
syndrome
osteoporosis
typical cervical spine are
C2-7
; atypical are
OA
+
AA
typical cervical spine:
upper cervicals tend to have more
rotation
+ less
side-bending
lower cervicals tend to have more
side-bending
+ less
rotation
C2-C7:
can be
neutral
,
flexed
, or
extended
somatic dysfunctions have
side-bending
+
rotation
to the same side
make up ~
50
% of flexion + extension of cervical spine
make up ~
50
% of rotation of cervical spine
OA:
~
50
% of flexion/extension of cervical spine
primarly
gliding
motion on cervical condyles
somatic dysfunctions found with flexion or extension + with
side-bending
+
rotation
to opposite sides
C1 tranverse process palpation:
posterior
to SCM
anterior
to angle of mandible
superior
by cartilage of auricle
ligaments that resist distraction also resist
flexion
of the CV joint
tectorial
membrane has been shown to be major restrain to distraction of the head from the
atlas
+
axis
anterior shear test for transverse ligament:
pt in
supine
anterior arches of
C2
are stabilized downwards towards
table
occiput
+
C1
are lifted using
lumbrical motion
pt instructed to count
backwards
outloud for
15
sec or until
end-feel
percieved
positive:
reproduction
of pt symptoms or
excessive
,
mushy end-feel
anterior stability test of
OA joint
:
pt in
supine
pads of fingers 2-5 cradle
occiput
pads of thumbs turned
medially
to fix the transverse mass of
atlas
+
axis
anterior
translation force applied
positive:
reproduction
of pt symptoms or empty
end-feel
brainstem S + S:
dizziness
drop
attacks
diplopia
dysphagia
dysarthria
ataxia
nausea
numbness
nystagmus
clinical CV instability frequently has
normal radiographic
findings
the transverse foramen contains:
vertebral
artery
sympathetic plexus
high risk factors for cervical instability"
>
65
years
dangerous
mechanism
paresthesia
in extremities
dangerous mechanisms include:
fall from elevation >
3
feet or
5
stairs
axial load
to head (i.e. diving)
MVC
high speed, rollover, or ejection
motorized
recreational
vehicle
bicycle
struck or
collision
posterior stability test of OA joint:
tests
transverse
lig
sides of cranium gentle compressed with
palms
of
both
hands
pads of
index
fingers over arch of
C2
- using
lumbrical
action - translate C2
anterior
under fixed
occiput
+ = excessive
anterior
translation of
C1
/
2
(relative
posterior
translation of occiput)
general (longitudinal) distraction:
tests
tectorial membrane
distraction performed with head + neck in
neutral
+ =
reproduction
of pt s + s
if negative, stress ligaments further by
biasing CV flexion
specific (longitudinal) distraction:
tests
AA membrane
C2 neural arch
fixed + CV flexed to eliminate
stabilizing
effect of
ligamentum nuchae
+ =
reproduction
of pt s + s
if the transverse ligament is torn, C1 will translate
forward
(
sublux
) on
C2
during
flexion
modified sharp-purser (safety test):
tests
transverse
ligament + subluxation of
C1
on
C2
pt in
sitting
CV
flexion
+ =
reproduction
of s + s or hearing/feeling a clunk
if negative, PT
stabilizes
C2 posteriorly + applies
posterior
force to
forehead
of pt
if the sharp-purser test is
positive
, the examination is
terminated.
the pt should be placed in a
cervical collar
and
emergency services
contacted
aspinall's test:
tests
transverse
lig
stabilize
occiput
on
atlas
in
flexion
hold occiput in
flexed
position
apply
anterior
force to
posterior
aspect of atlas
+ = pt feels lump in
throat
positive transverse ligament stress testing may result in:
muscle spasm
abnormally stretchy capsular end feel
production of
neurological symptoms
consistent
swallowing
during the test
transverse shear test:
tests
alar
lig
stabilize
mastoid
C1
moved in
transverse
direction
test repeated
stabilizing C1
+
translating mastoid
+ =
reproduction
of s + s;
empty end
feel
can also be performed on
C1
+
C2
alar ligament:
resists
contralateral side bending
stressed by passive
contralateral side bending
+
rotation
of occiput on
atlas
tension of alar lig during side bending results in
ipsilateral rotation
side
bending
stress test
alar lig
pt in supine
C2 neural arch
fixated with
mild compression
push pt right ear towards
left
side of
neck
(
SB right
)
minimal side bending occurs with
strong capsular
end
feel
+
solid stop
+ = empty end feel or
excessive translation
performed in neutral, CV flexion, + CV extension
kinetic test for alar lig:
pt in
sitting
C2
spinous process palpated for
motion
pt asked to
rotate
head
+ =
C2
spinous process does not immediately move when head is
rotated
(ligament
laxity
)
hypertonic
or
fibrotic rectus capitis posterior major
may give false negative for
alar
lig kinetic test
rectus capitis posterior major function:
extend
+
rotate
head
ipsilaterally
rotational stress test:
alar
lig
pt in
sitting
spinous
process +
lamina
of
C2
stabilized
head is
side
bent
C2 should immediately begin
rotating contralateral
to
side
bend
+ =
C2
does
not
immediately move
supine nodding test:
flexion
:
chin deviation indicates occiput is unable to glide on the side of deviation
extension
:
chin deviation indicates occiput is unable to glide on the opposite side of deviation
craniovertebral function:
make head movements
independent
from rest of vertebral column
orients the head in
3
planes
OA joint has 2 main functions:
transmit forces from
head
to
cervical
spine
provide significant degree of mobility in
sagittal
plane
AA rotation test:
pt in
supine
neck maximally
flexed
to chest - locks out
C2-7
pt
rotates
head
observe available
ROM
AA rotation ROM:
30-35
deg
ROM assessment of AA:
CV
side bending
head rotated in
opposite
direction