Save
...
Multi-Store Model
A03
Strengths
Save
Share
Learn
Content
Leaderboard
Learn
Created by
Kirsty T
Visit profile
Cards (25)
Based on a
lot
of
evidence
What are the other research to support the model?
Distraction
,
delay
and
interference
Brain damaged
patients
Primary
,
recency
and the
serial postion effect
What did Glanzer and Cunitz do?
Showed Ps
20
words in
succession
then asked to
free recall
What was the probability of recalling based on?
Where the
position
of the
word
was on the
list
(
serial position effect
)
What did the serial position effect show?
That people are
more
likely to remember words from the
beginning
and the
end
of the list than the
middle
What is the primary effect?
Remembering
words from the
beginning
What is the recency effect?
Remembering
words from the
end
of the list
What did the Brown-Peterson effect show?
That information from
STM
is
lost
if there's a
delay
between
memorising
information and
recalling
it
What % of information will be lost in 20s if there's no rehearsal?
90%
What are the causes of
forgetting
in STM

Lack of
capacity
What causes forgetting in LTM ?
Retrieval
failure,
decay
or
interference
What were the 2 main brain damaged paitients?
Clive Wearing
and
KF
What happened to CW?
Brain infection
caused by a
cold sore virus
How did this effect CW?
Destroyed
his
hippocampus
and parts of his
cortex
How did this effect CW?
His memory lasted about
7
seconds
He could still
walk
,
talk
and his
musical
ability was well preserved
His
capacity
of his
past
was extremely
patchy
What happened to KF?
Motorcycle
accident
What happened to his STM and LTM?
STM was
grossly
impaired
LTM was
normal
What was wrong with his STM?
Could only remember only
2
items or
chunks
How did KF disprove the MSM?
His LTM was
normal
which shouldn't be possible as if his STM was
reduced
than so should his
LTM
What was it?
Useful
,
simple
and
classic
model
What did it need?
Refining
not
rejecting
Why was it good as a first attempt to model memory?
Clear
and
easy
to
comprehend
Where did Atkinson and Shiffrin test their model?
In
lab
experiments and demonstrated
repeatedly
What did it show?
Clear differences
between these
memory
stores
What is an example of what is showed?
They differ in terms of
encoding
,
capacity
and
duration
The research in brain
damaged
patients supported the ideas of
3
memory stores