diaphragm flattens, increasing volume of thoracic cavity
as volume in thoracic cavity increases pressure decreases
Air moves down pressure gradient (from outside air, down trachea into lungs)
active process
Expiration:
external intercostal/ diaphragm muscles relax
ribcage moves down and in
diaphragm relaxes.... volume of thoracic cavity decreases.... pressure increases
Air moves down pressure gradient out of the lungs
passive process
alveoli adaptations:
thin walls- one cell thick... short diffusion pathway... faster rate of diffusion
large surface area- large number of alveoli... large surface area for gas exchange... faster rate of diffusion
steep concentration gradient-.....maintained by ventilation.... increases rate of diffusion
Tidal volume- volume of air in each breath
Ventilation rate- the number of breaths per minute
Forced expiratory volume (FEV)- the maximum volume of air that can be breathed out in 1 second
Forced vital capacity (FVC)- the maximum volume of air it is possible to breathe forcefully out of the lungs after deep breath in
Fibrosis:
formation of scar tissue in the lungs.... from infection/exposure to substances e.g. asbestos
scar tissue is thicker/less elastic than normal lung tissue
lungs... less able to expand... cant hold as much air as normal
Tidal volume/FVC reduced
diffusion is slower across thicker membrane
faster ventilation rate ... to get enough oxygen
Asthma:
smooth muscle lining bronchioles contracts/ large amount of mucus produced
constricts airways... airflow reduced
FEV reduced
Emphysema:
caused by smoking/ air pollution
causes inflammation... attracts phagocytes to area.... phagocytes produce enzyme which breaks down elastin
loss of elastin means alveoli cant recoil/ expel air as well... remain trapped in alveoli
also destroy alveoli cell walls.... reducing the surface area.... rate of gaseous exchange decreases
increased ventilation rate
TB
immune system builds a wall around the TB bacteria in lungs... forming small lumps... tuburcles
infected tissue within dies.. damaging gas exchange surface... decreasing tidal volume
causes fibrosis... further decreasing tidal volume
less air inhaled.... increased ventilation rate
Amylase (salivary glands/pancreas) catalyses hydrolysis of starch into maltose
hydrolysis of glycosidic bonds in starch
membrane-bound disaccharidases (enzymes attached to the cell membrane of epithelial cells) help break down disaccharides into monosaccharides (maltose->glucose)
monosaccharides can be transported across cell membranes via transporter proteins
monoglycerides/fatty acids + bile salts= micelles
Absorption/ transport of digested lipid molecules from the ileum into the lymph vessels:
Lipase hydrolyses ester bonds in triglycerides-> fatty acids/monoglycerides
Micelles contain bile salts + fatty acids/monoglycerides
Makes fatty acids/ monoglycerides more soluble in water
Bring/ release/ carry fatty acids/monoglycerides to cell/ lining of ileum
Fatty acids... absorbed by diffusion
Triglycerides reformed in cells
Vesicles move to cell membrane/exocytosis
Examples of xerophytic adaptations:
Stomata sunk in pits- traps moist air. reducing the conc gradient of water between leaf/air... reducing rate of diffusion out
'hairs' on epidermis- trap moist air around the stomata
Curled leaves with stomata inside- protecting them from wind (which increases the rate of diffusion)
reduced number of stomata
waxy, waterproof cuticles on leaves- reduce evaporation