Lecture

Cards (114)

  • Total lung capacity: amount of air that can be inhaled into the lungs on maximum inspiration
  • Tidal volume: amount of air inhaled and exhaled in 1 breath
  • Vital capacity: includes maximal inspiration and expiration
  • Residual volume: amount of air leftover after maximal expiration
  • Residual volume is very relevant to COPD because of air trapping that occurs
  • Ventilation contains 3 elements: neurological, innervation and diaphragm/intercostals
  • Sources of upper airway obstruction: FBAO, epiglottitis, burns and trauma
  • Source of lower airway obstruction: asthma and airway edema
  • Very rare to have to have lower airway burns but could be caused by certain gasses or chemicals
  • Things that interfere the alveolar membrane will also interfere with gas exchange
  • It takes about 1 minute for a RBC to circulate around the entire body so if O2 is applied, there should be a change within that time otherwise there's an issue
  • things affecting diffusion: inadequate O2(Histotoxic; CO poisoning/Cyanide), capillary bed pathology, severe atherosclerosis, alveolar pathology and interstitial space problem(Pulmonary edema or near drowning)
  • Saline doesn't have O2 in it so while it can help with blood pressure, it only does so much for SPO2 and maintaining perfusion
  • To build a differential Dx: think of anatomical components, think of worst first(Most life threatening/common) and think of less likely options as you work through assessments
  • Within chief complaint of SOB, consider word dyspnea, coughing(productive or nah) and frothy sputum
  • If there's no life threats, do an incident hx, focused assessment and then a history
  • In incident hx you're looking for: URI, colds, allergens from environment, cats/dogs, tobacco smoke, cold/dry air, exercise and GERD(gastric acid could cause bronchospasm)
  • Majority of respiratory problems are reactionary
  • Much harder to exhale in asthma
  • For PMHx in asthma, ask if they have a prior hx of asthma exacerbations, ask if they've been intubated before, ask if they've been hospitalized for it and ask about corticosteroid use(how recent and are they still on it)
  • On assessment for the head/neck: 1-2 word dyspnea, altered LOAs, head bobbing, central cyanosis, pursed lip breathing, tracheal tugging, JVD and retractions
  • On assessment for chest: symmetry, barrel chest, indrawing, retractions, accessory muscle use, positioning, depth/rate/regularity with breaths, chest auscultation and if coughing, is it productive
  • Generally chest auscultation can be done within the first 2 minutes of PT contact
  • Fine crackles occur in smaller airways and coarse on larger ones
  • Crackles are often heard on inspiration and usually bilateral in the lungs
  • Wheezes on inspiration is usually an obstruction like FBAO
  • Wheezes on expiration is usually asthma but it can also be on inspiration
  • Stridor is usually heard best over the neck
  • On assessment for extremities: ask for numbness, tingling, peripheral cyanosis and edema and colour/temperature/condition of the skin
  • On physical for pelvis: there could be incontinence caused by hypoxia
  • Seesaw breathing: type of abdominal breathing for kids in which when they inhale chest goes up and abdomen goes down and vice-versa for exhale.
  • Risk factors for exacerbation of asthma: gender(Females have more exacerbations), using 2 or more rescue inhalers per month, having a Hx of exacerbations, have other chronic health problems, smoker, not using meds as directed and having URI/Cold/Flu
  • Bradycardia in an asthmatic PT is an ominous sign of late stage hypoxia or an arrhythmia caused by the lack of O2
  • As air trapping increases in Pts, there's more pressure within the chest which could affect the hearts pumping ability.
  • Respiratory rate alone is not an indicator of distress
  • When bronchioles are normal, laminar airflow is present in which air flows without any resistance. Lack of laminar airflow increases resistance
  • ETCO2 has "sharkfins" in Asthma, COPD and CHF
  • Indications for PPV: Airway protection in a patient cannot maintain their own airways, hypercapnic respiratory failure, circulatory failure and poor tidal volume
  • Asthma is an air trapping disorder so PPV can cause them to fill up with air.
  • When PPVing asthma patients: reduce the volume of ventilations and ventilate at 8 to 10 breaths per minute