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Cards (45)
Functions of the Skin
Protects
against injury
Keeps
germs
out
Fluids
and
electrolyte
balance
Excretory
organ
Pain
perception
Vitamin
D
absorption
Maintains
body temp
Tissue destruction
Can have
local
and
systemic
effects
Sepsis
,
shock
, death
Infection prevention
is key
Closure of
burn wound
Systems affected by burns
Endocrine
Cardiac
Respiratory
Hematologic
Immune
Layers of the skin
Epidermis
-outer layer, it can grow back. No
blood
vessels
and has to get resources from the second layer of skin.
Dermis
-has
collagen
,
connective
tissue
,
elastic
fibers
,
blood
vessels
,
nerves
,
hair
,
lymph
vessels
, and
glands.
Subcutaneous
tissue - has a
basement
membrane
separating it from dermis
Dermis
Skin
regrowth
can occur as long as
parts
of the
dermis
remains
No
skin
regrowth
if the entire
dermal
layer
is
burned
and the cells and dermal appendages are
destroyed
Feet and hand have deeper dermal appendages which allows for
healing
of
deep burns
Nerves
Epidermis
burn is
painful
Dermis
is
painful
If a full
thickness
burn occurs the nerves may be
destroyed
, and
pain
may only be at the wound edges
May feel only
dull
or
pressure-like
pain
Burn classification by depth
Superficial-thickness-
only the
epidermis
(heals 3-6 days)
Partial-thickness-loss
of epidermis and part of dermis
Superficial partial
thickness- upper third of the dermis
Deep partial
thickness- deeper in the dermis, very few healthy cells remain
Full-thickness-
entire dermis and epidermis
Deep full-thickness-beyond
the skin into muscles bones and tendons
Type of Burn
Superficial
Superficial
partial
thickness
Deep
partial
thickness
Full
thickness
Deep
full
thickness
Electrical
Burn (A=entrance; B=exit)
Burn classification by injury
Mild-
treat and street
Moderate-
may need to go to burn center
Major-
need to go to a burn center
Rule of
9's
Fig
Vascular changes from burns
Excessive
weight
gain
w/i first
12
hours and continues
24-36
hours
Increased
macrophages
Blood vessel thrombosis
Fluid shift
Fluid
remobilization-
starts about 24 hours after burn
Hypokalemia and hyponatremia
Anemia
Continue to lose protein at wound site
Metabolic acidosis
Cardiac changes from burns
Hypovolemia
Dysrhythmia
Pulmonary changes from burns
Airway edema
Pulmonary capillary leak
Chest burns that restrict movement of chest
Carbon monoxide poisoning
GI changes from burns
Decreased
perfusion to
GI tract
Sympathetic nervous system causes
decreased motility
and even greater decrease in
blood flow
Curling's
ulcer
Metabolic changes from burns
Greatly
increased
Hypoglycemia
Increased
body temp
Always our jobs to
educate
patients ways to prevent
burns
Risk factors for death from burns
>
60
years old
>
40
% TBSA
Presence of
inhalation
injury
Goals of management in the 1st Resuscitation/Emergent Phase of Burn Injury
Secure
airway
Support circulation — fluid replacement
Prevent infection
Maintain body temp
Provide emotional support
General Management in Resuscitation phase
Airway patency
O2
as needed
Keep
warm
NPO
Elevate extremities
if no other obvious injuries
IV
with fluid replacement
Tetanus
toxoid
Head
to
toe
Respiratory
Assessment
Fire source, temp, environment, toxic, enclosed space?
Was
burn
to face, lips, ears, neck, eyes
Inspect
nose
, mouth,
pharnyx
Auscultate
lungs, bronchi, trachea
Inspect
chest
for
eschar
from circumferential burn
Review
carbon monoxide
poisoning
Cardiovascular assessment
Monitor for
hypovolemia
shock s/s
ECG
because of
electrolyte
imbalance or if electrical burn
Renal
assessment
Urine
output, color and consistency, debris
BUN
/Crea,
NA
GI assessment
Motility
/ bowel sounds/ occult blood in any stool or vomit
>
25
% TBSA get NG tubes
Skin Assessment
Size
and
depth
of injury
BSA
affected
Laser Doppler
image to determine depth
Lab values to review
H/H
BUN
GLU
Electrolytes
T.protein
,
alb
ABG
Xrays
CT
, US,
Bronchoscopy
, MRI
Nursing
diagnosis
Interventions to support pulmonary functioning
Airway
maintenance
Positioning
,
deep
breathing
Promote
ventilation
Monitor for
gas exchange
O2 therapy
Antibiotics
, diuretics, lol's,
paralytics
Preventing hypovolemic shock
1.
Rapid fluid infusion- Parkland formula 4ml X kg
X
TBSA
(This is times not divided by.)
2.
1st 24 hour dosing
give ½ the first 8
hours
and then the 2nd ½ over next 16 hours
3.
Monitor
for s/s of shock
Surgical Management of Burns in Emergent Phase
Escharotomy
Fasciotomy
(Done when burnt to the bone)
Pain management
Opioids-
know what route is best to administer and why
Complementary and alternative options
Environmental
changes
Surgical
excision
Preventing
respiratory
distress
Wound care nonsurgical management
Remove
exudates
and
necrotic
tissue (debridement)
Clean
Stimulate
granulation
and
revascularization
Dressing
Silver Nitrate
Apply with a
gauze
dressing
Reduces fluid
evaporation
Bacteriostatic
Inexpensive
But it does penetrate the
eschar
and it can
deplete
Na and K
Silver sulfadiazine
Apply with a clean
glove
Effective against both gram neg/pos and yeast
But it can cause
neutropenia
,
decreases
granulocyte formation
Penetrates
eschar
some
Discoloration a
gray
or
blue-green
Mafenide acetate
BID
Penetrates
eschar
and
bacteriostatic
But it is
painful
to apply and remove
Can cause
metabolic acidosis
Nonsurgical debridement
Mechanical: use
scissors
and forceps to cut away the dead tissue during
hydrotherapy
Chemical: Apply a topical enzyme (
collagenase
) to the wound during
daily
dressing changes
Dressing the Burn Wound
Standard
wound dressings-
distal
to proximal
Biologic
dressings-
temporary
Biosynthetic
dressings- commonly used on
partial
thickness burns
Synthetic
dressings-clear,
decrease
pain, low infection rate, reduced cost
Surgical management
Grafting-
autografting
Xenograph
Nursing care of grafts
Maintain
immobilization
of graft sites
Elevate
extremities
Monitor the graft for
infection
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