Uses evidence from the crime scene and other details to fit into pre-existing categories of organised or disorganised offender
Organised offender
Crime tends to be planned
Victim is specifically targeted
Maintains high degree of control during crime
Leaves little evidence
Generally high in intelligence, socially and sexually competent, live with partner, have car, follow crimes in media
Disorganised offender
Crime tends to be unplanned
Random selection of victim
Very little control during crime, impulsive
Body/evidence left at crime scene
Tend to have lower than average IQ, in unskilled work or unemployed, history of sexual dysfunction and failed relationships, live alone
Top-down profile construction
1. Data assimilation
2. Crime scene classification
3. Crime reconstruction
4. Profile generation
Bottom-up approach
Data-driven using statistical databases, grounded in psychological theory
Investigative psychology
Matches crime scene details with statistical analysis of typical offender behaviour
Focuses on interpersonal coherence, time/place, forensic awareness
Geographical profiling
Studies spatial behaviour to infer offender's home/operational base
Canter's circle theory - marauder vs commuter
Monoamine oxidase A (MAOA) gene
Gene that produces an enzyme that breaks down neurotransmitters like dopamine and noradrenaline, linked to aggression
MAOA-L
Low activity form of MAOA gene, results in less enzyme production and higher neurotransmitter levels, linked to aggression and offending behaviour
Genetic transmission of criminal behaviour
Criminal behaviour runs in families and is transmitted genetically
Twin studies show higher concordance rates for identical twins vs non-identical twins
Monozygotic (MZ) and dizygotic (DZ) twins
MZ twins are identical, DZ twins are non-identical
If MZ twins have higher concordance rates for offending behaviour than DZ twins
Genetic factors are involved in offending behaviour
Lange (1930) study
Investigated 13 MZ and 17 DZ twins where one twin had served time in prison
Found 77% concordance rate for MZ twins and 12% for DZ twins
Concluded genetic factors play a predominant part in offending behaviour
Genetic approach to offending behaviour
Suggests offending behaviour has a genetic basis
Supported by twin studies showing higher concordance rates for MZ twins compared to DZ twins
The gene for MAOA (monoamine oxidase A) is linked to aggression and offending behaviour
Brunner et al. (1993) study
Investigated a Dutch family where several generations of men had a history of aggressive and violent acts
Found the men had a genetic condition (Brunner syndrome) resulting in lower intelligence and MAOA deficiency
Offers evidence that individuals can inherit genetic conditions that make them prone to offending behaviour
Christiansen (1977) found concordance rates of 33% for MZ twins and 12% for DZ twins, suggesting offending may have a genetic component
A problem with twin studies is that most twins are reared in the same environment, so concordance rates may be due to shared learning experiences rather than genetics
Diathesis-stress model
Suggests offending behaviour arises from the combination of genetic predisposition and environmental factors (e.g. dysfunctional upbringing, criminal role models)
Caspi et al. (2002) study
Found low activity version of MAOA gene alone was not a significant predictor of aggression, but low MAOA gene plus maltreatment had a significant effect
Genetic explanations are criticised as biologically deterministic, suggesting genes completely control behaviour
Evidence suggests genetics alone cannot fully explain offending behaviour, as not everyone with a 'criminal gene' becomes an offender
Neural explanations of offending behaviour
Focus on differences in brain structure and neurotransmitter levels
Suggest offending is linked to aggression and impaired impulse control
Amygdala
Part of the limbic system that processes emotional information, implicated in offending behaviour as it is involved in the stress response and aggressive impulses
Prefrontal cortex
Regulates impulse control and social interactions, damage has been linked to antisocial and offending behaviour
Serotonin
Regulates mood and impulse control, low levels could be linked to offending behaviour
Noradrenaline
Abnormally high levels have been associated with aggression, violence and criminality
Raine et al. (2000) found reduced brain activity in areas like the prefrontal cortex and abnormalities in the limbic system in offenders compared to controls
Strengths of neural explanations
Use of objective scientific methods like PET scans
Potential for practical applications like dietary interventions to alter neurotransmitter levels
Neural explanations are reductionist, simplifying offending behaviour to biological factors and overlooking social/environmental factors
Atavistic form
Lombroso's theory that criminals have distinguishing physical features indicating they are 'genetic throwbacks' to a more primitive stage of evolution
Lombroso's findings on physical features of different types of criminals
Used a scientific, evidence-based approach to study criminals
Raised the possibility of scientific study of the criminal mind
Goring (1913) failed to replicate Lombroso's findings when comparing convicts and non-convicts
Lombroso's theory was criticised as biologically deterministic and socially sensitive/racist
Eysenck believed criminals have a personality type characterised by high extraversion, neuroticism and psychoticism
Extraversion
Extraverts are sociable, impulsive, expressive and risk-taking, and need more external stimulation
Antisocial behaviour
Behaviour that goes against social norms and expectations, often associated with criminal behaviour
Even though it is a psychological theory of personality, Eysenck believed that each trait has a biological basis which is mainly innate and comes about through the type of nervous system we inherit