Produce sebum through hair follicles, which make skin oily
Prevent water loss
Sweat glands
Eccrine - smaller, coiled tubules which open to skin surface and located over the entire skin
Apocrine - larger, open to hair follicles, located mainly in axillae, areolae of the breasts and genital area, produce thick secretions which react with bacteria on skin surface to produce body odor
Subcutaneous Layer (Hypodermis)
Consists mostly of fat
Provides protection, insulation, and caloric source
Hair
Composed of keratin
Can be fine (vellus hair) or darker and thicker (terminal hair)
Nails
Composed of keratin
Clear with highly vascular bed of epithelial cells underneath
Infants
Lanugo - fine soft hair present at birth
Skin is thinner, less fat - more prone to dehydration and hypothermia
Pregnancy
Linea nigra - line down midline of abdomen
Chloasma - face of pregnancy
Striae gravidarum - stretch marks
Aging
Stratum corneum thins, loss of collagen, elastin, and fat, decrease of sebaceous and sweat glands
More prone to dehydration and hypothermia
History of skin disease - what was it? how was it treated? does it run in the family?
Significant familial predispositions - allergies, hay fever, psoriasis, eczema, acne
Use of nonsterile equipment for tattoos increases risk of Hep C
Change in pigmentation might suggest systemic illness (jaundice)
Change in a mole
Pruritus - any dryness? is it seasonal? (xerosis - dry, seborrhea - oily)
Excessive bruising - consider abuse, frequent minor trauma may be sign of alcohol abuse
Rash or lesion - onset, location, spread, character or quality, duration, associative factors, alleviating and aggravating factors, patient's perception
Medications - prescription and over-the-counter, may indicate allergy to medication
Hair loss or growth - gradual or sudden, hirsutism - unusual growth
Change in nails
Exposure to hazards - environmental or occupational, bitten by bee, tick, mosquito? exposure to plants or animals?
Self-care - what cosmetics, soaps, chemicals? possible allergies
Physical Examination - Color
General pigmentation should be even throughout
Benign pigmented areas - freckles (macules) on sun exposed skin, nevi (moles) - junctional, compound, dysplastic, birthmarks
Vitiligo - absence of melanin in patchy areas
Pallor
Pale, white color caused by decrease of blood flow (vasoconstriction) or decrease in hemoglobin
Brown skinned people will be more yellow, black skinned people will be more gray
Palpebral conjunctiva and nail beds should be observed
Caused by shock, anemia
Erythema
Redness due to increased blood flow (vasodilation)
If fever suspected, check skin for warmth. If edema, check skin for tightness
May be caused by fever, inflammatory process, emotions, CO poisoning
Cyanosis
Bluish, purplish hue due to decreased perfusion of tissues
Darker skinned people have normal bluish tone on lips, palms, but not clearly evident, other clinical signs should be observed
May be caused by hypoxemia due to heart failure, shock, chronic bronchitis
Jaundice
Yellow, orange hue due to jaundice (increased bilirubin in blood)
Hard and soft palate must be observed in addition to sclera of eyes
Dark urine also present
Due to liver problems such as hepatitis, cirrhosis
Temperature
Check skin with dorsa of hands, hyperthyroidism may cause increase of temp
Moisture
Diaphoresis may occur during fever or exercise
Dehydration can be observed by dry mucous membranes in mouth and cracked skin
Mobility and Turgor
Mobility is ease of skin rising when pinched, turgor is returning back to its place
Slow turgor can be indicative of dehydration, "tenting" if severe dehydration
Lesions
Any traumatic or pathological change in skin
Roll nodule gently between fingers to assess depth
Ultraviolet light is used if fungal infection suspected (Wood's light)
Edema
The presence of excess interstitial fluid; an area that appears swollen, shiny and taut and tends to blanch the skin color or, if accompanied by inflammation, may redden the skin
It may also described as pitting or non-pitting edema
Pressure Ulcers
Injuries to the skin and underlying tissue, primarily caused by prolonged pressure on the skin
They can happen to anyone, but usually affect people confined to bed or who sit in a chair or wheelchair for long periods of time
Nursing Responsibility for Pressure Ulcers
1. Use repositioning schedules (every 15 minutes when on chair or every 2 hours when on bed)
2. Use pressure mattress or chair cushion
3. Use lifting devices as directed to reduce shear (trapeze bar for patients, or lifts for family, if necessary)
4. Use positioning with pillows or wedges to avoid bony prominence contact with surfaces and to maintain body alignment
5. For those who are bedbound, avoid elevating the head of bed beyond 30 degrees except for brief periods