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PDTCP
Tx Planning and Clinical Practice
ANUG
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Daisy Robb
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Cards (29)
what are the classification of acute periodontal disorders?
gingivitis
or
periodontitis
what are the classifications of
gingivitis
?
traumatic
chemical
bacterial
viral
fungal
classifications of
periodontitis
?
abcess
traumatic
/
parafunctional
bacterial
acute inflammation
is
sudden
in onset and
short
in durations. Will see:
erythema
,
swelling
,
pain
,
warmth
,
loss
of
function
NUG is a
destructive
disease of the
periodontium
caused by plaque bacteria and is confined to the gingival tissues
AKA: Acute
necrotising
ulcerative
gingivitis
(ANUG), Trench
mouth
, Vincent’s Infection
Prevalence of NUG?
0.001
% in british population
17-25yrs
higher in those with
HIV
high in
malnourished
children following
viral
/
protozoal
infection
aetiology?
opportunistic
infection of
gingival
tissue by
commensal
organisms
fusospirocheatal
complex
spirochaetes
invade tissue first
fusiform
bacillus
mixed
anaerobic
infection
Microbiology?
prevotella
intermedia
fusobacteria
selmonas
treponema
Why use
USS
?
water
disrupts
biofilm
physically
oxygen
bubbles burst releasing
oxygen
, killing
anaerobes
pathogenic mechanism of NUG?
spirochaetes
and
fusiforms
can invade tissue by
direct
toxic
effects
indirect activating
and
modifying
host response
develops in association with
predisposing
factors which
lower
body's defenses
systemic predisposing factors of NUG?
smoking
stress
fatigue
diet
immunocompromisedd
local predisposing factors of NUG?
smoking
pre-existing perio
/
gingivitis
poor oh
clinical features of NUG?
painful bright red gingiva
bleeding
tenderness
on probing
halitosis
ID papilla
ulcerated
ulcer
covered with
yellow
/
grey slough
submandibular lymphadenopathy
high
temp?
what is slough?
pseudomembranous
first stage of NUG:
tip of id
papilla
in col
common
lower
ants
crater-like
defects
second stage of NUG:
gingivae
bleed readily
halitosis
mod pain
initially
third stage of NUG:
necrotising
lesions develops
rapidly
pain more
severe
yellow
/
white
/
grey
slough
pronouce
halitosis
fourth stage of
NUG:
ulceration
may spread to adjacent
tissues
pseudomembranous
covering
lymphandentitis
raised
temp
crater-like
defect
punched
out
papilla
initial
stage of NUG
moderate
NUG
more progressed
NUG
how would we treat the
acute phase
?
debridement:
removing gross deposits
of
plaque
and
calculus
using
LA
,
USS
flushes, removing
sloughing material
to
reveal
ulcerated
bleeding tissue.
personal home care plan:
intensive OHI
,
mouthwash
and
stop smoking
systemic antibiotics
review app 3-5 days later to review OHI and continue scaling
what type of MW recommended?
peroxyl
-
oxidising
mw
3
%
hydrogen peroxide
chx
mw
0.2
% x
10ml
bd for
1
min
what antibiotics might be prescribed?
metronidazole 200mg
-
400mg
tds
3
days (
pregnancy
/
warfarin
?)
penicillin
V
250mg
qds
5
days
long term tx?
improve
OH
scaling
smoking
cessation
diet
advice
stress
management
surgical
correction
/recontouring
counselling
for
recreational
drugs
Why might recurrence occur?
after
acute
phase subsides,
inadequate
tx and predispoding factors
persists
NUG
necrotising ulcerative periodontitis
necrosis
of
periodontal ligaments
and
bone
NUP
NUS -
necrotising ulcerative stomatitis