nerve that carries sensory information from the face to the brain
trigeminalnuclei
cranial number 5 (trigeminal)
pons
responsible for consciousness
locked-in syndrome
lesion in pons occulomotor
lower pons
upper pons
RESPIRATION
Pneumotaxiccenter
promotes expiration and controls rate and depth of respiration, upper pons
apneusticcenter
inspiration
basal ganglia is part of the extrapyramidaltract
thalamus is relay station between sensory input from spinal cord to cortex
MEDULLA OBLONGATA
primary respiratory center; involuntary respiration; vaso motor center (BP and heart rate); emetic center (vomiting center); coughing reflex, sneezing and yawning
RIGHTC4
right side of the body have motor loss and Fx; right motor propriception and leftpain and temp
DIRECT PATHWAY
agonist (prime motor) GPI
ANATOMICAL
caudate nucleus
putamen
globus pallidus
FUNCTIONAL
straitum
subtansya nigra pars compacta
THALAMUS
sends signals
BROADMANAREA4
receives stimulation and sends motor signals to corticospinal
INDERECTPATHWAY
antagonist; does not fascilitates movement in would inhibit
CHOREA
rapid jerks
ATHETOSIS
SLOWMOVEMENT
Spinal cord pathways
Dorsal column-medial lemniscus pathway
Cuneate fasciculus
Fasciculus gracilis
Spinocerebellar tracts
Posterior spinocerebellar tract
Ventralspinocerebellar tract
Anterolateral spinothalamic tracts
Lateral spinothalamic tract
Anterior spinothalamic tract
Spino-olivary tract
Pyramidal tracts
Lateral corticospinal tract
Anterior corticospinal tract
Extrapyramidal tracts
Reticulospinal tract
Rubrospinal tract
Vestibulospinal tract
Olivospinal tract
Brown-Séquard syndrome
Below the lesion: ipsilateral motorparalysis; ipsilateralloss of proprioception, twopointdiscrimination and vibrationsense; contralateralloss of pain and temperaturesensation.
Anterior cord syndrome
Below the lesion: bilateral motor paralysis; bilateral loss of pain, temperature and light touch sensation; bilateral preservation of proprioception and vibration sensation.
Posterior cord syndrome
Below the lesion: intact motor function; bilateral loss of pain of proprioception, vibration, fine touch and two-point discrimination sensation.
Central cord syndrome
Below the lesion: bilateral motor paralysis with sacral sparing and lower limb weakness < upper limb weakness; bilateral loss of pain, temperature and light touch sensation
Afferent (input) fibers to the striatum
Corticostriate
Thalamostriate
Nigrostriate
Other
Corticostriate fibers
Origin: Sensory cortex (Brodmann's areas 3, 1 and 2), Primary motor cortex (Brodmann's area 4), Premotor cortex (Brodmann's area 6), Supplementary motor cortex (Brodmann's area 6), Frontal eye field association areas of cerebral cortex
Termination: Putamen
Neurotransmitter: Glutamate (excitatory) or Aspartate (excitatory)
Thalamostriate fibers
Origin: Intralaminal nuclei of the thalamus, Centromedian nucleus, Parafascicular nucleus, Ventral anterior and ventral lateral nuclei of the thalamus
Termination: Caudate nucleus, Caudate nucleus and putamen
Composed of two cerebellar hemispheres and the intervening vermis
Surface displays alternating slender parallel elevations (ridges) known as folia and depressions (grooves) known as sulci
Connected to the dorsal aspect of the brainstem by three pairs of prominent fiber bundles, the superior, middle, and inferior cerebellar peduncles
Has an outer rim of gray matter, the cortex, an inner core of nerve fibers, the medullary white matter, and the deep cerebellar nuclei located within the white matter
Cortex and white matter are easily distinguished in a midsagittal section, where the white matter arborizes, forming the core of what appears to be a tree-like architecture, known as the arbor vitae
Cortex is a three-layered structure: molecular layer, Purkinje layer, and granular layer