SA node dysfunction (sinus bradycardia, sinus pause/arrest)
AV node dysfunction (first degree heart block, second degree heart block (Wenckebach, Mobitz type 2), third degree heart block (complete))
On an ECG, we pay particular attention to p waves, PR interval and QRS complex
First degree heart block
Prolonged PR interval
Second degree heart block (Wenckebach or Mobitz Type 1)
Progressive prolongation of PR interval followed by a dropped QRS complex
Second degree heart block (Mobitz Type II)
Constant PR interval followed by a droppedQRS complex, QRS complexes appear in clumps, more likely to cause hemodynamic compromise and progress to complete heart block than Mobitz 1, needs pacemaker
Third degree heart block
No relationship between P waves and QRS complexes, needs pacemaker
Management of bradycardia
1. Assess if patient is stable or unstable
2. Seek and treat reversible causes
3. Monitor the patient
4. Provide O2 for hypoxia
5. Address ischemia and hypotension
6. Identify and treat any underlying systemic conditions e.g. electrolyte disturbances
First line medication for bradycardia
Atropine (0.6mg, maximum of 3mg)
Second line medications for bradycardia
Isoprenaline infusion
Adrenaline infusion
Life-threatening causes of bradycardia
Hypoxia
Heart failure
Severe lung diseases
Approach to treatment of bradycardia
1. Medical management (medications like pacing agents, vasodilators, inotropes)
If the causes of bradycardia are treatable, targeting them is the primary approach used to manage the condition
Tachycardia
Rapid heart rate
Approach to tachycardia
1. Don't panic
2. Ask yourself: narrow or wide, regular or irregular, presence/absence of P waves, timing of P waves relative to QRS complex
3. Assess condition of patient (stable or unstable)
Amiodarone
A class-III antiarrhythmic drug that works by blocking sodium channels, reducing depolarization rate and decreasing heart rate. It also acts on calcium channels to further slow depolarization and prolong repolarization.
Severe lung diseases
Certain severe lung diseases, such as COPD or pneumonia, can cause a lack of oxygen in the body and lead to bradycardia
Ventricular tachycardia
Ectopic cells within the ventricles stimulating the heart to contract
Causes of ventricular tachycardia
Myocardial muscle deterioration
Drug stimulation
Symptoms of Unstable ventricular tachycardia
Change in mental status or loss of consciousness
Managing stable ventricular tachycardia
Antiarrhythmic medications- Amiodarone 300mg IV over 10-20 mins, then 900mg over 24 hrs
Managing unstable ventricular tachycardia
Immediate cardioversion
What to do after converting ventricular tachycardia to a sinus rhythm
Administer an antiarrhythmic infusion- Amiodarone 300mg IV over 10-20mins, then 900mg over 24 hrs
Apply the management principles of ventricular tachyarrythmias
1. Assessment
2. Stabilization
3. Medical management
4. Cardioversion for severe tachycardia
Assessment
The first step is to assess the patient's condition and the situation. This includes monitoring vital signs and performing a thorough physical examination.
Stabilization
Stabilizing the patient includes providing oxygen, maintaining airway clearance, and controlling bleeding, if any.
Medical management of VT
Medical management involves giving medications like anti arrhythmic drugs, vasodilators, sedatives, and electrolytes.