Systolic blood pressure greater than 180 mmHg or less than 90 mmHg
Oxygen saturation less than 90% despite supplementation
Acute change in mental status
Urine output less than 50 ml over 4 hours
Staff member has significant concern about the patient's condition
Preventing pressure injuries
1. Determine risk level
2. Skin care bundle - STAND
3. Nutrition
4. Fluid intake
5. Documentation
Reducing pressure
1. Head of bed
2. Positioning
3. Source of pressure
4. Standing
5. Pressure-offloading devices
6. Specialized beds
7. Turning schedule
Common dressing techniques
Wet-to-damp: saline moistened gauze
Continuous wet gauze
Topical enzyme preparations
Moisture-retentive dressing
Nursing care for wounds
1. Dressing changes
2. Pad contact surfaces
3. Use lift sheet
4. No rubber ring or donut on sacral area
5. Pillow or foam wedges between bony prominences
6. Keep skin off plastic surfaces
7. Skin care
8. Nutrition: fluids- 2-3 liters daily and 1.25-1.5 grams/kg of protein
Pain
What the patient says it is
Types of nociceptive pain
Visceral: pain from organs
Somatic: pain from skin, muscle, tendons
Referred: u have an injury, but u feel pain somewhere else
Radiating: pain travels to another area in body
Neuropathic pain
Originates from nerve injury, pain continues even after the painful stimuli is gone, sensations may include numbness, tingling, burning, aching, crushing, stabbing, or shooting
Psychogenic pain
Pain perceived by a person when there is no physical cause for pain, caused, increased, or prolonged by mental, emotional, or behavioral factors
Pain assessment
Provocative or palliative
Quality or quantity
Region or radiation
Severity scale
Timing
Non-opioid considerations
Monitor for hepatoxicity (acetaminophen)
Monitor for gastric side effects (NSAIDs)
NSAIDs carry a risk for cardiovascular and renal adverse effects through prostaglandin inhibition
Opioid antagonists
Naloxone, naltrexone, no analgesic effect
Adverse effects of opioid analgesics
Respiratory depression (less common)
Hypotension
Constipation (common)
Nausea
Vomiting
Pruritus
Sedation
Aldosterone
Adrenal cortex secretes, prevents water & sodium loss, triggers kidneys to reabsorb sodium & water from urine into the blood, increases blood osmolarity & volume, promotes kidney potassium excretion
Antidiuretic hormone (ADH) (vasopressin)
Hypothalamus signals posterior pituitary gland to release ADH, ADH travels to the kidneys and retains only water, indirectly regulates electrolyte retention or excretion
Natriuretic peptide (NP)
Secreted with increased blood volume and pressure, stretches heart tissue, binds to receptors in nephrons, opposite effect of aldosterone, urine output is increased, decreased blood volume & decreased blood osmolarity, kidney reabsorption of Na+ inhibited
Causes of hypokalemia
Inadequate intake of dietary potassium
Potassium-wasting drugs, such as the loop diuretic furosemide (Lasix)
NPO status
IV fluids without supplemental potassium
Certain medications (corticosteroids and chemotherapeutics)
Signs and symptoms of hypokalemia
Dysrhythmia
Hypoactive bowel sounds
Constipation
Bilateral muscle weakness
Flaccid paralysis
Respiratory Depression
Interventions for hypokalemia
1. Monitor ECG
2. Monitor laboratory values
3. Dietary Intake of Potassium
4. Oral Supplementation
5. IV Supplementation SAFE PRACTICE ALERT: Never administer IV potassium as a push or bolus medication
Causes of hyperkalemia
Impaired renal excretion
Excessive intake of potassium
Certain medications, such as potassium- sparing diuretics
Cushing syndrome
Extensive tissue damage resulting from trauma or burns
Severe infections causing the release of intracellular potassium
Signs and symptoms of hyperkalemia
Dysrhythmia, including bradycardia and heart block
Transient abdominal cramping
Bilateral muscle weakness
Rare to have respiratory involvement
Flaccid paralysis
Cardiac arrest
Interventions for hyperkalemia
1. Monitor vital signs
2. Monitor ECG
3. Monitor lab data, including potassium, BUN, and creatinine
4. Oral drug patiromer
5. Avoid potassium-rich foods
6. Administer IV fluids including glucose and insulin
Signs and symptoms of hypocalcemia
Increased Neuromuscular excitability
Positive Chvostek sign and Positive Trousseau sign
Numbness and tingling of extremities
Muscle cramps that can progress to tetany
Hyperactive reflexes
Laryngospasm
Seizures
Sources of calcium
Cheese
Ice cream
Spinach
Rhubarb
Yogurt
Milk
Tofu
Interventions for hypocalcemia
1. Monitor vital signs
2. Monitor ECG
3. Implement fall/seizure precautions
4. Dietary Intake of Calcium
5. Calcium supplements
6. Administer oral/IV calcium supplements (lactated ringers) as ordered
Causes of hypercalcemia
Hyperthyroidism
Cancer
Vitamin d toxicity
Signs and symptoms of hypercalcemia
Decreased Neuromuscular excitability
Anorexia
Nausea and vomiting
Constipation
Personality change
Lethargy
Stupor (low LOC)
Coma
Decreased muscle strength and tone
Interventions for hypercalcemia
1. Monitor vital signs
2. Monitor ECG
3. Increase oral fluid intake to 3 L for adults
4. Increase patient activity
5. Decrease intake of calcium-rich foods
Signs and symptoms of hyponatremia
Malaise
Anorexia
Decreased level of consciousness
Confusion
Lethargy
Coma
Seizures
Interventions for hyponatremia
1. Determine Cause
2. Monitoring lab data
3. Monitoring intake and output
4. Administering hypertonic IV solution
5. Restricting water in the diet
6. Adding table salt to foods
Signs and symptoms of hypernatremia
Thirst
Decreased level of consciousness
Confusion
Lethargy
Coma
Interventions for hypernatremia
1. Drug therapy- Furosemide or bumetanide
2. Nutrition therapy
Osmolarity
Normal value = 270-300 mOsm/L, Dehydration= 300<, Overload= <270
Dehydrationlabs
Hemoglobin and hematocrit
Serum osmolality
BUN and creatinine
Urine specific gravity
Urine osmolality
Fluid overload labs
Serum electrolytes
Hematocrit
B U N
Serum osmolality
Albumin
Types of IV solutions
Isotonic = 270 to 300 mOsm/L
Hypertonic = Fluids >300 mOsm/L
Hypotonic = Fluids <270 mOsm/L
Chronic kidney disease (CKD)
Progressive and irreversible disorder, declines gradually, diagnosed when the GFR drops below 60 mL/min/1.73 m2 for more than 3 months, most common cause of death is cardiovascular disease
Fluid and electrolyte imbalances in CKD
Hyperkalemia- kidneys lose the ability to excrete potassium
Sodium levels vary widely due to fluid volume changes
Hypermagnesemia- caution use of milk of magnesium, magnesium citrate and antacids containing magnesium
Hypocalcemia
Elevated Phosphorus
Metabolic Acidosis
Hematological and respiratory signs/symptoms in CKD
Anemia, Bleeding Tendencies, Infection
Kussmaul breathing, which results in increased CO2 removal by exhalation, Dyspnea may occur as a manifestation of fluid overload, pulmonary edema, uremic pleuritis (pleurisy), pleural effusions, and respiratory infections (e.g., pneumonia)