central nervous system includes the brain(non-myelinated) and spinal cord (myelinated )
peripheral nervous system is composed of the afferent (sensory) and efferent (motors) system.
efferent system is composed of the somatic and autonomic system
somatic system is conscious reflexes, myelinated. There is one neurone between the CNS and effector. The effector is skeletal.
autonomic system is involuntary movements, non-myelinated and usually 2 or more neurones between CNS and effector. This system can be split into the sympathetic and parasympathetic systems.
sympathetic system speeds things up and employs flight or fight response. Uses noradrenaline and also long post-ganglionic neurones so meaning multiple effectors and shortpre-ganglionic neurones.
parasympathetic system slows things down, uses Ach. Has few long pre-ganglionic neurones and short post-ganglionic neurones
to increase heart rate, the medulla oblongata sends an action potential via accelerans which releases noradrenaline which binds to receptors on SAN so increase heart rate.
to decrease heart rate, medulla oblongata sends an action potential ia vagus nerve which releases Ach which binds to nicotine receptors on SAN which decrease heart rate
heart rate can also increase if adrenaline binds to receptors on SAN
endocrine system involves the hypothalamus, pituitary gland, adrenal cortex and thyroid gland
sliding filament theory is how the actin and myosin slide over each other to make the muscles contract
myosin is a fibrous protein with globular heads. The heads bind to actin-myosin active sites, they are hinged and contain ATPase
actin is a globular protein made of many actin subunits ; tropomyosin, troponin and actin
troponin has three subunits that bind to ; calcium, tropomyosin and actin
tropomyosin covers the binding sites at rest
sliding filament
when muscle fibre depolarises calcium leaves the sarcoplasmic reticulum and binds to troponin
this causes tertiary structure of troponin to change which pulls tropomyosin out of way revealing binding sites
myosin heads attach and from cross-bridge
myosin heads move actin over myosin = power stroke
an increase in calcium stimulates ATPase which breaks ATP into ADP+Pi which releases energy to break the cross-bridge
myosin heads returns to original shape and then attaches to another actin binding site and process is repeated many times
ATP uses in sliding filament theory
break cross-bridge
ATP pumps calcium back into sarcoplasmic reticulum
at the neuromuscular junction
Action potential arrives at the junction and voltage gated calcium channels open so calcium enters
vesicles containing Ash move to and fuse with membrane releasing Act into self via exocytosis
Ach diffuses across cleft and binds to Ash nicotinic receptors and sodium then enters the muscle fibres
if get to -55mv, voltage gates sodium channels open = depolarisation.
depolarisation causes calcium to leave the sarcoplasmic reticulum
examples of responses to biotic factors :
alkaloids = made from amino acids and have bitter taste
tannins = found throughout plant with horrid taste, binds to proteins in gut in herbivores so they can't digest protein and in insects they are toxic
pheromones = released by one organism that cause a response in another organism
two types of growth response : tropism and nastic
tropism is directional and examples include : phototropism, geotropism, chemotropism, thigmotropism, hydrotropism and thermotropism
nastic growth is non-directional growth and examples are thimonasty
auxins have four main functions ;
cause apical dominance
causes positive phototropism in shoots and negative phototropism in roots
causes positive geotropism in roots and negative e geotropism in shoots
inhibits leaf loss (abscission) in deciduous plants
causes apical dominance
apical bud/tip produces IAA which stimulates growth of apical tip
IAA then diffuses down and inhibits growth in lateral shoots
in talks plants IAA at bottom is low so lateral shoots may grow here
causes positive phototropism in shoots
IAA binds to receptors and hydrogen ions are pumped from cytoplasm to cell walls
pH decrease and stimulates enzymes to break hydrogen bonds in cellulose and increase hydrogen ions and decrease water potential causing water to enter
causing the cell to elongate
causes negative phototropism in roots
IAA produced in root
IAA acclimates on shady side
and inhibits cell elongation so root bends away from light
causes positive geotropism in roots
IAA produced by root tip
IAA accumulates on underside and inhibits cells elongation
root then bends down
causes negative geotropism in shoots
IAA is produced in bud
IAA accumulates on underside and stimulates cell elongation
so shoot bends up
evidence of geotropism
happens in dark
put in klinostat which spins seedlings and inhibits effect of gravity roots/shoots grow horizontally
put shoots and roots in Petri dishes at different angles and all the shoots will bend up and roots will bend downwards
inhibits leaf loss in deciduous plants
happens to prevent excess water loss as water is frozen
leaf loss is triggered hen day length decrease and light intensity decreases
in spring young leaves produce auxins to inhibit leaf loss and cytokinins
cytokinins inhibit senescence and stimulate cell division and help shoots and roots develop quicker in tissue culture and also stimulate lateral shoot formation
in autumn cytokinin levels drop which causes senescence, causes auxins levels to drop ad cause ethylene to increase which stimulates cellulase to breakdown cells in the abscission layer meaning leaves fall off
ethylene causes fruits and flowers to ripen by positive feedback
gibberellins are produced by young leaves and seeds and stimulate seed germination, elongation, lateral shoot formation and flowering
when seeds absorb water in spring they produce gibberellins which stimulate amylase and protease production causing growth
to make gibberellins a precursor and an enzyme are needed
abscisic acid inhibits seed germination and growth and stimulate stomatal closure when less water is available
ABA
when water levels are low, ABA binds to receptors on guard cells which causes calcium to leave vacuoles and enter cytoplasm
this causes ion channels in cell wall to open and potassium to leave guard cell
this decreases water potential outside guard cels and so water leaves by osmosis and guard cells become flaccid and stomata close