Use for muscle grade 0-1 (from no muscle contraction to visible or palpable muscle contraction)
Functional Electrical Stimulation (FES)
Use for muscle grade 2-3 (from full range of motion against gravity eliminated to full range of motion against gravity)
Effects of Physical Agents
Inflammation and healing
Pain
Collagen extensibility and motion restrictions
Muscle tone
Process of healing
1. Inflammation
2. Proliferation
3. Maturation
Inflammatory Phase
Lasts for 1 to 6 days, cells that remove debris and limit bleeding enter the traumatized area, characterized by heat, swelling, pain, redness, and loss of function
Goals of treatment during Inflammatory Phase
Prevent further injury (bleeding)
Clean open wound
PRICE
Protection
Rest
Ice
Compression
Elevation
POLICE
Protection
Optimal
Loading
Compression
Elevation
Chronic Inflammation Phase
First 3 days - 20 days, collagen is deposited in the damaged area to replace tissue that was destroyed
Goals of treatment during Chronic Inflammation Phase
Prevent or decrease joint stiffness
Control pain
Increase circulation
Maturation/Remodelling Phase
Last phase, damaged collagen is now replaced, formation of hard callus
Goals of treatment during Maturation/Remodelling Phase
Regain or maintain strength
Regain or maintain mobility
Control scar tissue formation
Pain
Controlled by modifying pain transmission or by changing the underlying process that is causing the sensation
Ways to control pain
Modulating spinal cord level transmission
Changing the rate of nerve conduction
Altering the central or peripheral release of neurotransmitters
Motion Restriction
Agents can be effective adjuncts to the treatment of motion restrictions caused by muscle weakness, pain, soft tissue shortening, or a bony block
Modalities for certain cases of motion restriction
Muscle weakness - ES/FES
Bony Block - Shock wave
Spastic patient - Thermotherapy
Hypotonic muscle - Cryotherapy
Tone Abnormalities
Agents can alter muscle tone directly by altering nerve conduction, nerve sensitivity, or biomechanical properties of muscle or indirectly by reducing pain or the underlying cause of pain
General contraindications and precautions
Pregnancy
Malignancy
Pacemaker or other implanted electronic device
Impaired sensation
Impaired mentation
Pregnancy
Effects of energy produced by modalities on fetal development usually are unknown
Malignancy
Physiological effects may reach malignant tissue or alter the circulation to such tissue, potentially accelerating growth or metastasis
Pacemaker or other implanted electronic devices
May alter the functioning of the device
Impaired sensation and mentation
Patient may be unable to properly/accurately report sensation
Considerations in selecting a physical agent
Goals and effects of treatment
Contraindications/precautions
Evidence
Cost, convenience, availability
Pain Control Theories
Melzak and Wall's Gate Control Theory
Endogenous Opiates
Central Biasing
Melzak and Wall's Gate Control Theory
Ascending mechanism: Increased non-nociceptive stimuli from periphery into spinal cord = pain relief; Receptors from A-beta fibers can decrease the input of nociceptive stimuli from continuing to the second-order neuron
Gate Control Theory: Opening and closing the gate
Interaction of nonpain-carrying fibers (A-beta) and nociceptors (C fibers and A-delta) at secondary afferent neurons; More active nonpain tract = closing the gate on pain transmission
Large and small afferent interaction
The tract with the most activity will prevail and be allowed to continue up the spinal cord; The speed of transmission has nothing to do with opening and closing of the gate