host cell, infected by bacteria, displays antigens on its MHC and becomes an APC
T-killer cells with complementary receptors bind to the APC
cytokines, previously secreted by T-helper cells, stimulate T-killer cells to divide by mitsosis forming active T killer cells and T memory cells
active T-killers biding to infected cells becoming APC
T-killers release chemicals, which form in infected cells causing lysis- T memory remain to allow immediate SIR to occur if reinfected