cardiovascular control centre located in medulla oblongata
controls the rate SAN generates electrical impulses
these impulses cause atria to contract thus determines rhythm of heartbeat
changes act as stimuli
stimuli detected by baroreceptors(found in aortic and carotid bodies) and chemoreceptors(located in medulla oblongata and A/C bodies)
baroreceptors detect:blood pressure
chemoreceptors detect: PH, C02 levels, 02 levels
neurones involved
once receptors are stimulated, send impulses to medulla which sends impulses to SAN along sympathetic or parasympathetic neurones- release different neurotransmitters which affect SAN in different ways
sympathetic= increase rate SAN generates electrical impulses(increases HR)
parasympathetic= decreases rate SAN generates electrical impulses(decreases HR)
changes in HR- blood pressure
high blood pressure CCC=cardiovacular control centre(in medulla)
detected by baroreceptors-CCC- impulse sent along parasympathetic neurones-acetylcholine(NT)
NT binds to receptors on SAN causing it to fire less frequently=reduced heart rate and BP back to normal
changes in HR-blood pressure
low blood pressure
baroreceptors detect-impulses to CCC-impulses sent along sympathetic neurones- noradrenaline released(NT)
NT binds to receptors on SAN causing it to fire more frequently
Heart rate increases and blood pressure increases back to normal
changes in HR- blood conc.
high blood 02, low c02 or high PH
chemoreceptors detect changes- sends impulses to CCC-sends impulses along parasympathetic neurones-acetylcholine released
NT binds to receptors on SAN
causing it to fire less frequently, HR slows and blood levels return to normal
changes in HR- blood conc.
low 02, high C02 , low PH
chemoreceptors detect changes- send impulses to CCC-impulses sent along sympathetic neurones- noradrenaline released
NT binds to receptors on SAN causing it to fire more frequently
HR speeds up and blood chemical levels return to normal