light enters the eye via the pupil and is focused onto region of retina called fovea
amount of light entering eye is controlled by muscles in iris
cilary muscles attached to lens via suspensory ligaments
fovea has many light and photoreceptors
photoreceptors
rod cells
outer retina
sensitive to light intensity
only black and white images
cone cells
grouped together in fovea
sensitive to different wave lengths of light so detects colour
can be red,green or blue sensitive-images in colour
action potentials generated in photoreceptors are sent to the brain via optic nerve
optic nerve leaves via the blind spot(no receptors present)
photoreceptors+nerve impulses
generate AP when stimulated by bright enough light(rods)or particular wavelengths(cones)
light sensitive pigments in photoreceptors are bleached when light falls on them
rhodopsin
rod cells contain light senstive pigment RHODOPSIN
when light hits rhodopsin it breaks into retinal and opsin
this breaking apart=bleaching- causing a chemical change in photoreceptors and results in generation of nerve impulses
nerve impulse-bipolar neurone-opticnerve-brain
the action of rod cells
rod cells initiate an AP in neighbouring bipolar neurones when they are hyperpolarised
in the dark:
1)NA+ is actively pumped out of rod cells creating a concentration gradient
2)NA+ diffuses back down this concentration gradient and into rod cells via sodium channels- rod cell is depolarised
3)depolarised rod cell released an inhibitory neurotransmitter which diffuses across synapse to the bipolar neurone
4) NT inhibits depolarisation in bipolar neurone, no generation of AP=preventing a nerve impulse being sent to optic nerve
in the light
1)light bleaches rhodopsin=retinal and opsiN
2)bleaching causes NA+ channels in surface membrane of the rod cells to close- preventing sodium ions from diffusing back into the rod cell as opsin binds to membrane
active transport of NA+ out is still taking place so NA+ removed and not returned
3)lack of + ions in rod cell causes interior to become more negative until it hyperpolarises
4)hyperpolarised rod cell stops releasing inhibitory NT so generation of AP in neighbouring bipolar neurone no longer inhibited
5)AP generated in bipolar neurone- impulse sent to optic nerve