based on knowledge that certain NTs are connected to the condition
Abnormal levels of NTs thought to cause condition - moderating levels using drugs
Anti-depressants normally used - SSRIs
Mode of action
SSRIs effect at synapse
Blocking reuptake transporter for serotonin on pre-synaptic axon terminal
Serotonin remains longer in synapse - increased opportunity to bind at receptor sites
Alternative drug therapies
other antidepressants like SNRIs or Anxiolytic drugs - facilitating brains main inhibitory NT -GABA
Strength 1
P - research in support from Soomro (2009)
E - meta-analysis of 17 studies that compares SSRIs to placebos for treatment - all studies showed SSRIs moreeffective - 70% symptoms declines other 30% responded to alternative drug therapy
E - use of drugs to address biochemical unbalances successful and improved wellbeing of people with OCD
L - more valid and appropriate treatment to offer
Strength 2
P - more accessible and costeffective that therapies
E - drugs are readily available to treat OCD and much cheaper for public health services
E - don't require effort/commitment from patient and not disruptive to their lives - take drug until symptoms declines
L - more people feel encouraged/ have ability to use drug therapy
Weakness
P - associated sideeffects
E - indigestion, tremors
E - affect compliance with drugs - can lead to return of symptoms - reduction in wellbeing
L - costs need to be carefullyweighed against benefits to choose to offer