Perio basics

• Gingiva is part of masticatory mucosa that covers the attachment apparatus.
• Gingival margin should be scalloped in form and firmly attached.
• The distal wedge procedure is performed in the areas distal to the oral cavity.
• Gingival tissue should be resilient and fibrotic in nature.
• Osteoplasty is used to re-shape or re-contour the bone.
• The flap design in periodontal surgery is determined by the location of the surgery.
• The "work-horse" of periodontal surgery is the flap design.
• Osteoplasty will not provide attachment for the periodontal fibers.
• The distal wedge procedure is used to reduce excess tissues that provide access for underlying bone.
• Stipplings of the attached gingiva are present.
• Results from the absence of stipplings include edema of the underlying connective tissue, inflammation of the gingival collagen, and normal anatomy with two parts (macroscopic).
• Oral epithelium is lined with keratinized, stratified squamous.
• Periodontal probing is used to assess the amount of attachment loss.
• Physiologic tooth mobility allows accommodate masticatory forces.
• Being is the most accurate for bone loss assessment.
• Prognosis is a questionable term.
• CADIA (Computer-Assisted Densitometric Image Analysis) is used to assess periodontal disease progression.
• A DNA Probe identifies a particular microorganism present in the plaque.
• Pathologic tooth mobility results from loss of connective tissue attachment.
• Peri-apical is the most common method for preliminary assessment.
• Miller's Classification of Tooth Mobility Grade I: Horizontal mobility < 1mm Grade II: 1mm Grade III: > 1mm and Vertical Mobility.
• Glickman Classification of Progression Grade I: No bone loss Grade II: Partial bone loss Grade III: Total bone loss.
• Radiographs are used to assess the amount of bone destruction.
• Pocket depth measures the distance from the gingival margin to the deepest probing depth.
• Hopeless periodontitis is defined as more than 5mm clinical attachment loss.
• Sulcular epithelium is lined with non-keratinized, stratified squamous.
• Biologic width is formed by the junctional epithelium, which is attached via hemidesmosome to the lamina propia.
• Junctional epithelium contains basket cells (10-29), mostly found near the sulcus.
• CLINICAL SIGN of acute necrotic gingivitis is bleeding on probing due to an increased LYMPHOCYTES in the gingival sulcus with FEVER that lasts for more than 3 days.
• AGGRESSIVE PERIODONTITIS is characterized by bone loss in patients aged 12-25 years old.
• GONADOTROPHIC HORMONE feeds to increased Prevotella intermedia and Capnocytophagia species.
• AGGRESSIVE PERIODONTITIS mainly affects rapid, severe bone loss with a VERTICAL PATTERN.
• Prevotella intermedia is a sex steroid-induced microorganism.
• CLINICAL SIGN of periodontitis is the elaring of anterior teeth.
• ACUTE NECROTTING GINGIVITIS can occur in the developing period (4-1 days) with ulcerations, collagen destruction via COLlagenase, and blood circulation.
• RADIOGRAPHIC SIGN of periodontitis is AIVEOAR BONE LOSS.
• PT BERTAL (MENSTRUAL) GINGIVITIS is caused by hormonal imbalance associated with GOOD ORAL HYGIENE.
• PERIODOITIS is the inflammation of periodontium with PERIODONTITIS is usually preceded by GINGIVITIS, and can progress to periodontitis.
• TREATMENT for acute necrotic gingivitis includes antibiotics and antiseptic mouthwash.
• PREGIITANCY TUTOR can lead to a severe granulomatous inflammation during the period of gestation.