Action potentials is generated at the axon hillock. The RMP much reach a threshold of -55mV
Neurotransporter binds to sodium or chloride channels which will increase or decrease the charge
Excitation postsynaptoc potentials os EPSP increase the likelihood of an action potential from occurring by increasing the value and getting closer to the threshold
Inhibitory synaptic potentials or IPSPS decrease likelihood of action potential by decreasing the charge. Located along initial segment
UPSO and ESPSP are added in the axon hillock to determine mV
In the initial segment, sodium voltage gated channels are opened and once 30mV is reached, sodium channels will deactivate and will open potassium channels to repolorize the cell. The cell will reach -80mV and will slowly return at RMP during the state of hyperpolarization
In the conductive segment the action potential will open sodium channels to depolarize the cell until 30mV. Potassium channels then open up and bring the charge down in the process called repolarization. Potassium remain open longer than needed and slowly go up to MRP in state called hyperpolarization
In the refractory period, the time to start action potential is either difficult or impossible
In the transmission segment, the signal can run either continously or saltatorily
In the transmission segment, the change in V will open calcium channels to let them in and bind to vehicles. The calcium will push the vesicle to the synaptic know end and push it out via exocytosis. Calcium pumps will then pump calcium out and restores calcium concentration outside the wall