The International Association for the Study of Pain (IASP)
has defined pain as “an unpleasant sensory and emotional
experience, which we primarily associate with tissue damage
or describe in terms of such damage” (IASP, 2011). The most
important definition of pain as it is experienced is that by
McCaffery and Pasero (1999): “Pain iswhatever the person
says it is.” It is important to remember this definition when
assessing and treating pain.
Recent literature has emphasized the importanceand under-treatment of pain, and has recommended that pain be the fifth vital sign
Some states have passed laws necessitating the adoption of an assessment tool and documenting pain assessment in client records along with temperature, pulse, heart rate, and blood pressure
The Joint Commission has established standards for pain assessment and management
Inadequate treatment of acute pain has been shown to result in physiologic, psychological, and emotional distress that can lead to chronic pain
Healthy People has added a new topic for 2020 that includes pain as it affects "Health-Related Quality of Life and Well-Being"
Health-related quality of life (HRQoL)
A multidimensional concept that includes domains related to physical, mental, emotional and social functioning. It goes beyond direct measures of population health, life expectancy and causes of death, and focuses on the impact health status has on quality of life
Pain
A combination of physiologic phenomena but with psychosocial aspects that influenceperception of thepain
Pathophysiology of pain
1. Transduction
2. Transmission
3. Perception
4. Modulation
Nociceptors
Sensory receptors that detect signals from damaged tissue and chemicals released from the damaged tissue
Located at the peripheral ends of both myelinated nerve endings of type A fibers or unmyelinated type C fibers
Three types: mechanosensitive,temperature-sensitive, and polymodal
Nociceptors are distributed in the body, skin, subcutaneous tissue, skeletal muscle, joints, peritoneal surfaces, pleural membranes, dura mater, and blood vessel walls, but not located in the parenchyma of visceral organs
Transduction of pain
1. Noxious stimulus results in tissue injury or damage stimulating the nociceptors
2. Inflammatory process leads to release of cytokines and neuropeptides
3. Activation of primary afferent nociceptors (A-delta and C fibers)
delta primary afferent fibers
Small-diameter, lightly myelinated fibers that transmit fast pain to the spinal cord within 0.1 second, felt as pricking, sharp, or electric-quality sensation, usually caused by mechanical or thermal stimuli
C fibers
Unmyelinated, primary afferent fibers that transmit slow pain within 1 second, felt as burning, throbbing, or aching, usually caused by mechanical, thermal, or chemical stimuli resulting in tissue damage
Transmission of pain
1. Inflammatory process results in conduction of impulse in primary afferent neurons to the dorsal horn of the spinal cord
2. Neurotransmitters released and concentrated in the substantia gelatinosa
3. Output neurons from the dorsal horn cross the anterior white commissure and ascend the spinal cord in the anterolateral quadrant in ascending pathways
The anterolateral tracts relay sensations of pain, temperature, nondiscriminative (crude) touch, pressure, and some proprioceptive sensation
The process of pain perception is still poorly understood
Emotional status (depression and anxiety)
Affects directly the level of pain perceived and thus reported by clients
Hypothalamus and limbic system
Responsible for the emotional aspect of pain perception
Frontal cortex
Responsible for the rational interpretation and response to pain
Modulation of pain is a difficult phenomenon to explain
Modulation of pain
1. Changes or inhibits the pain message relay in the spinal cord
2. The descending modularly pain pathways either increase (excite) or inhibit pain transmission
Endogenous neurotransmitters involved with modulating pain
Endogenous opioids, such as endorphins and enkephalins
Serotonin
Norepinephrine (noradrenaline)
Gamma-aminobutyric acid (GABA)
Neurotensin
Acetylcholine
Oxytocin
Pain elicits a stress response in the human body that triggers the sympatheticnervoussystem
Physiologic responses to pain
Anxiety, fear, hopelessness, sleeplessness, thoughts of suicide
Focus on pain, reports of pain, cries and moans, frowns and facial grimaces
Decrease in cognitive function, mental confusion, altered temperament, high somatization, and dilated pupils
Increased respiratory rate and sputum retention, resulting in infection and atelectasis
Joint Commission Standards for Pain Management
Require health care providers and organizations to improve pain assessment and management for all patients
Patients have a right to
Appropriate pain assessment and management
Pain assessment and management
1. Screen initially
2. Assess periodically (nature and intensity)
3. Record pain assessment results
4. Follow-up with reassessments
Assess staff for
Level of knowledge in pain assessment and management
Educate staff
In pain assessment and management as needed
Organizational policies and procedures
Support appropriate ordering or prescribing of effective pain medications
Discharge planning process
Address patient needs for symptom management
Collect data
To monitor the appropriateness and effectiveness of pain management
Pain classification
Duration
Location
Etiology
Severity
Types of pain based on duration and etiology
Acute pain
Chronic nonmalignant pain
Cancer pain
Acute pain
Usually associated with a recent injury
Chronic nonmalignant pain
Usually associated with a specific cause or injury and described as a constant pain that persists for more than 6 months
Cancer pain
Often due to the compression of peripheral nerves or meninges or from the damage to these structures following surgery, chemotherapy, radiation, or tumor growth and infiltration
Pain location classifications
Cutaneous pain (skin or subcutaneous tissue)
Visceral pain (abdominal cavity, thorax, cranium)
Deep somatic pain (ligaments, tendons, bones, blood vessels, nerves)
Pain location
Perceived at the site of the pain stimuli
Radiating (perceived both at the source and extending to other tissues)
Referred (perceived in body areas away from the pain source)