1. statistical infrequency - the more we see something, the more it is likely to be normal. any unusual behaviour is abnormal
2. deviation from social norms
3. failure to function adequately - a person finds it difficult to complete the demands of everyday life. Rosenhan and Seligman - three signs: no longer conforming to standard interpersonal rules, severe personal distress, behaviour becomes irrational.
4. deviation from ideal mental health. Marie Jahoda- criteria: rational and able to perceive oneself accurately, self actualisation, can cope with stress etc.
- one strength is that there are real life applications, has important implications for therapies and explains why patients need to be exposed to the fear stimulus, preventing avoidance reduces reinforcement so fear declines, can be used to improve a patients' life
- one limitation is that operant conditioning cannot explain the maintenance of some phobias, evidence to suggest that some avoidance behaviours is motivated by positive feelings of safety e.eg only being able to leave the house with a trusted person, theory cannot be generalised to all phobias, lowers explanatory power of the explanation
- one limitation is that there is an opposing theory, sometimes phobias do not follow traumatic experiences and are just scared of snakes or spiders, it can be passed down through genes maximising our chance of survival in the world
- one strength is that there is research evidence to support it, Gilroy et al followed up on 42 patients who had been treated for spider phobia through systematic desensitisation, they were less fearful than the relaxation group who were taught relaxation without exposure, effective in unlearning the maladaptive response through counter-conditioning
- one strength is that SD is suitable for a diverse range of patients, flooding is not ideal for some patients, some have learning difficulties and may find it hard to relax in extreme situations, SD useful as the process is broken down
- one limitation is that flooding can be traumatic for patients, patients may give consent but many are unwilling to see it to the end, they are using time effort and money to not get rid of the phobia and could cause psychological harm
- one limitation is that flooding is less effective for some phobias, for complex phobias like social phobias , there are cognitive aspects which link more to unpleasant thoughts about the situation
Compulsions - extreme anxiousness or uncomfortable feeling due to an unwelcome stimulus (repetitive and reduce anxiety), Avoidance - attempt to reduce anxiety by staying away from potential triggers
- Family studies - lewis states that 37% of OCD patients has parents and 21% had siblings with ocd, OCD runs in families but it is more about genetic vulnerability not certainty, diathesis stress model shows that some people are more likely to suffer OCD as some environmental stresses are necessary to trigger it
- candidate genes - creates a vulnerability for oCD, some of these genes are involved in regulating the development of the serotonin systematic. e.g. 5HTI-D beta plays a role in the transport of serotonin across synapses
- OCD is polygenic - caused by many genes
- different types of OCD - one group of genes may cause OCD in one person but a different group of genes may cause the disorder in another person - aetiologically heterogeneous
- the role of serotonin - if a person has low levels of serotonin , normal transmission of mood relevant information will not take place, resulting in mental processes being affected.
- decision making systems - impaired decision making may be associated with abnormal functioning on the lateral of the frontal lobe, the left parahippocampal gyrus (associated with the processing of unpleasant emotions) also functions abnormally in OCD
drug therapy - increases or decreases levels of neurotransmitters in the brain, low levels of serotonin are associated with OCD
- SSRIs - selective serotonin reuptake inhibitor is most commonly used as an antidepressant for OCD, it works by blocking reuptake allowing more serotonin to pass messages between neurons (pre-synaptic neuron -> post-synaptic neuron)
- alternatives to SSRIs - where an SSRI is not effective after 3-4 months, it can be combined with other drugs such as tricyclics (same effect, more side effects) or SNRIs (increase levels of serotonin and noradrenaline)
- combining SSRIs with other treatments - Drugs are often with cognitive behavioural therapy to treat OCD, they reduce emotional and behavioural symptoms allowing them to engage more effectively
- one strength is that there is supporting evidence, Nestadt et al found that 68% of identical twins shared OCD as opposed to 31% non-identical twins, evidence that some people are more vulnerable to OCD due to genetic makeups
- one weakness is that there are too many candidate genes, psychologists have been much less successful at pinning down all the genes involved, cannot be certain which variations are responsible for OCD
- one strength is that the treatments can be provided, antidepressants increasing serotonin levels are effective in reducing OCD symptoms, suggest that the serotonin system is involved in OCD, treatments are useful in treating OCD
- one strength is that there is research evidence to support it, Soomro et al reviewed studies that compared SSRIs to placebos and all 17 studies showed significantly better results, symptoms declined by around 70%, effective
- one strength is that it is not expensive, drug treatments are more accessible to the public, provided by NHS in the UK, can be used alternatively to expensive methods like CBT
- One limitation is that there are severe side effects, research found that those who take clomipramine, more than 1 in 10 suffer tremors and weight gain, more than 1 in 100 become aggressive and suffer disruption to blood pressure and heart rhythm, can cause discomfort that is not beneficial to the patient.